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After completing my undergraduate and postgraduate studies at the University of Melbourne, I had a Postdoctoral position at the University of Melbourne from 2011-2013 and held a Victorian Postdoctoral Resarch Fellowship at Kings College London (UK) from 2014-2015 and University of Melbourne in 2016. I took up an Australian Research Council DECRA Fellowship at Monash University from 2017-2019 and was a National Imaging Facility research fellow from 2020-2021. I joined The University of Queensland in 2022 as the Head of Radiochemistry at the Centre for Advanced Imaging.

Ralph Patrick is a researcher focussed on understanding the molecular drivers of ageing and age-associated diseases and developing new therapeutic approaches to help alleviate diseases of ageing. He is trained as a computational biologist, with a BSc (Hons) and PhD from the University of Queensland (UQ). After completion of his PhD in 2016, he worked as a postdoctoral scientist at the Victor Chang Cardiac Research Institute (VCCRI) in Sydney for nearly six years. At the VCCRI, a major focus of his research was mapping out how the individual cells of the heart respond to a heart attack at the gene expression level and how these compare to other forms of chronic heart disease. Following the VCCRI, he joined the Ageing and Cellular Reprogramming lab at the IMB in 2022 as a postdoctoral fellow. His work at the IMB focusses on understanding the epigenetic and transcription factor drivers of the ageing process and leveraging this knowledge to develop new strategies for restoring youthful cell states. Any potential collaborators or students interested in this research area are welcome to contact him.

Dr Omkar Nadh Pattela is a Research Fellow at the University of Queensland, based at the Law School and affiliated with the Centre for Policy Futures. His research spans science and technology studies (STS), public health, and regulatory governance, with a focus on how institutional, political, and economic dynamics shape biomedical innovation.

He is currently undertaking a qualitative project on synthetic biology as part of the ARC Centre of Excellence in Synthetic Biology. This research explores how earlier controversies surrounding genetically modified organisms (GMOs) have influenced the governance, regulation, and public legitimacy of emerging synthetic biology applications.

He recently completed a multi-year study on the invention and commercialisation of the HPV vaccine, analysing how patent disputes, university–industry partnerships, and licensing practices shaped its development and global access. The project contributes to broader debates on intellectual property, academic capitalism, and the political economy of health technologies.

Dr Pattela earned his PhD in Development Studies from the Institute for Social and Economic Change, India. His doctoral research examined the political economy of medical biotechnology in India’s life sciences sector and was published as a monograph by Routledge in 2022. With interdisciplinary training in systems biology, STS, and intellectual property law, he brings a cross-sectoral lens to both research and teaching. He has taught at institutions in India and Australia on innovation policy, responsible science, and development studies.

Dr Cassandra Pattinson research centres around exploring the effects of sleep and circadian rhythms on health, wellbeing, and recovery across the lifespan. Dr Pattinson is a Senior Research Fellow at the Child Health Research Centre (CHRC), an ARC Discovery Early Career Award (DECRA) Fellow, and a Senior Research Fellow as part of the the ARC centre of Excellence for the Digital Child. Her work has been supported by the ARC, NHMRC, NIH and the DSTG, as well as the Australian Federal Government and Queensland Government.

Her research has involved a range of populations from children and adolescents, through to military personnel and athletes. Dr Pattinson's research spans a range of study designs and methodologies, including longitudinal studies tracking large child cohorts (>2000 children), standard observation techniques, survey and individualised standard child assessment, as well as studies employing physiological (actigraphy, spectrometry) and biological (hormones, proteomic, genomic) designs. Dr Pattinson also has a strong track record in research translation, these have included manuscripts in top scientific journals, reports for government and non-government organisations, development of professional development programs, as well as designing and presenting vodcasts and resources (e.g. fact sheets, workshops) to parent groups, young adults, government departments and the early childhood sector.

At CHRC Dr Pattinson is a part of the Community Sleep Health Group. This group collaborates with many other groups around broader issues of sleep and technology, sleep and the environment (including disasters), mental health and wellbeing, pain, disability, and new technologies and approaches.

• Director, Department of Nuclear Medicine & PET Services, Royal Brisbane & Women’s Hospital
• Theranostics Lead, Department of Nuclear Medicine & Specialised PET Services, Royal Brisbane & Women's Hospital
• Co-Director of Training, Department of Nuclear Medicine & Specialised PET Services, Royal Brisbane & Women’s Hospital
• Professor, School of Medicine, University of Queensland

David underwent dual specialty training in both Endocrinology and Nuclear Medicine at Royal Melbourne Hospital, Austin Hospital and Peter MacCallum Cancer Centre in Melbourne. He was appointed a staff specialist at Peter MacCallum Cancer Centre at the completion of his training, where he continued to gain experience in a quaternary referral centre for theranostics treating patients from across south-east Australia and New Zealand. In 2016 he moved to Royal Brisbane & Women’s Hospital where he is currently the Director of Department of Nuclear Medicine & PET Services and nuclear medicine lead for the RBWH radionuclide therapy service.

Julie’s research is mainly focussed on gas-water-rock core reactivity at reservoir conditions using experimental, field, and geochemical modelling techniques. Recent projects have been in the application of carbon dioxide geological storage in which CO2 is captured and stored in formations generally contained by low permeability cap-rock. The safe containment of the injected CO2 and the potential changes to rock porosity, permeability, and water quality should be determined. Recent and current projects with a focus on a demonstration site in the Surat Basin (Precipice Sandstone) include the impacts of impurity or acid gases present in industrial CO2 streams (collaboration with D. Kirste, SFU), inducing carbonate precipitation (in collaboration with S. Golding), and understanding dissolved metal sources and fate. Julie has also worked closely with the CO2CRC, CTSCo, Glencore, SEAL, the NSW government, CI-NSW, and ANLEC R&D, and provided expert opinion to the Queensland Government, and input to Environmental Impacts Assessments.

Julie is currently working with landholders, the QLD regional government, RDMW, councils and industry to understand the sources of methane in aquifers of the Great Artesian Basin, especailly those overlying coal seam gas reservoirs (CSG) (with Arrow Energy, SANTOS, APLNG, H. Hoffman, K, Baublys).

Other projects include gas-water-rock or acid-rock reactivity that modify nano-porosity and gas flow in gas or oil bearing shales.

Julie Pearce graduated with an MCHEM (Hons) degree in Chemistry from the University of York, UK. She then moved to the University of Bristol to complete a Ph.D. in 2007 focusing on laser spectroscopic studies to understand the detailed reaction dynamics of atmospheric processes. From 2007 – 2009 she accepted a Japan Society for the Promotion of Science Postdoctoral Fellowship, hosted at Nagoya University, Japan. There she measured delta 13C and delta 18O isotopic signatures of CO2 simultaneously in real time in the atmosphere using a laser spectroscopic technique to understand anthropogenic and biogenic sources of CO2. After taking a career break to travel in 15 countries in Asia, she moved to Brisbane in 2010 where she is enjoying the surrounding natural beauty of Queensland.

See Google Scholar page

Research achievements other than research outputs

Dr Pedroso is an expert in numerical and computer methods for solid mechanics and materials modelling. He has a strong background in tensor calculus, partial differential equations, computational geometry, and computer programming, among other topics. Dr Pedroso has been developing methods to model the mechanical behaviour of porous media including mixtures of solids, liquids and gases. Dr Pedroso has also developed new methods in molecular dynamics to model solids and granular assemblies. Therefore, his research work is quite multi-disciplinary but revolves around computational engineering and mechanics.

Dr. Pedroso received the highly prestigious Argyris Lecture Award of 2016 from the University of Stuttgart, Germany, which is internationally recognized. The Argyris Lecture is the top award for experts working on Modeling and Simulations, in particular, with the Finite Element Method (FEM), because Prof Argyris is a pioneer of the FEM. Today, this method is the most attractive for approximating the solution of partial differential equations with complex geometries and boundary conditions. One key aspect that the committee considered in the award was the innovative papers on new techniques for porous media, such as a new method to handle unilateral and variable boundary conditions for the interface between liquid and gases within porous media.

Journal Reviews

Dr Pedroso is an expert in computational mechanics for porous media and optimisation and is reviewing papers for top journals such as Computer Methods in Applied Mechanics and Engineering, International Journal for Numerical Methods in Engineering, International Journal for Numerical and Analytical Methods in Geomechanics, Nature: Scientific Reports, Computers and Geotechnics, Geotechnique Letters, Advances in Engineering Software, Journal of Engineering Mechanics ASCE, Computer Physics Communications, International Journal of Plasticity, Soils and Foundations, Advances in Structural Engineering, Engineering Structures, among others.

Research Grants Reviewer

Dr Pedroso is an Australian Research Council (ARC) reviewer for DPs, DEs and LPs. He is also a reviewer for th Hong Kong Research Grants Council HK-RGC

Conference Services

Dr Pedroso has organised the 1st Workshop on New Advances on Computational Geomechanics in Australia in 2008 and the 5th Workshop on New Frontiers in Computational Geotechnics in 2010. Both in Brisbane, Australia.

Dr. Andre A. Pekerti is an Associate Professor in International Management within the International Business Discipline at The University of Queensland Business School. He is an n-Cultural: a Christian of Indonesian-Chinese heritage who grew up in Jakarta, Southern California, and New Zealand, and is a naturalised New Zealander and Australian. Andre’s multicultural background complements his research interest and teaching in international management.

His primary research topics are attributions, acculturation, cultural intelligence, cross-cultural communication, multi-method assessment on n-Culturals and cognitive complexity, ethics, family business, human factors, n-Culturals, servant leadership, social justice, trustworthiness, and wellbeing.

Andre consistently publishes in international journals including, International Journal of Cross-Cultural Management, International Journal of Human Resource Management, Journal of Business Ethics, International Journal of Intercultural Relations, and Journal of International Business Studies.

He served as Vice President of the Australia New Zealand International Business Academy for two terms. He consistently serves as an ad hoc reviewer for several international journals. Currently, he is on the following editorial review boards: Asia-Pacific Journal of Management; Cross-Cultural and Strategic Management; Human Resource Management Review; International Journal of Intercultural Relations; Journal of International Business Studies; Journal of World Business; and Honorary Editor of Andalas Management Review.

Andre initiated the Rapid Acculturation Mateship Program (RAMP) at UQBS. A precursor to Global Mates and BEL Buddies, RAMP is a 19-week program connecting local students with incoming international students. RAMP “Serves to facilitate adjustment to UQ, Australia and the reciprocal learning of cultures”, and has positively affected the experiences of domestic and international students.

Dr. Pekerti has taught in MBA Programs at The University of Auckland, Bond University and UQ Business School. He consulted for Diner’s Club, Singapore; the Department of Transport Victoria, Australia; and the Ministry of Trade, Indonesia. Most recently, I collaborated with BiasSync, a science-based solution designed to help organisations more effectively assess and manage unconscious bias in the work environment.

Author of n-Culturalism in Managing Work and Life: A new within individual multicultural model.

My research interests have concentrated on the molecular genetic analysis of multigene phenotypes of bacteria encompassing pathogenicity, bacterial degradation of synthetic environmental pollutants, photosynthesis and the synthesis of antitumour antibiotics. My PhD research focussed on plasmids and mapping of the genome of the human pathogen P. aeruginosa (Pemberton,and Holloway, 1972a; Pemberton,and Holloway,1972b;Pemberton and Holloway,1973). I continued this research as a postdoc at UC Berkeley with John Clark in the Department of Molecular Biology in the Wendell Stanley Virus laboratory. I am grateful to Mark Guyer who taught me how to isolate large plasmid DNAs. In Robley Williams lab I learnt how to use the Kleinschmidt and Zahn technique for spreading the plasmid DNA on an electronmicroscope grid and metal shadow the sample to visualise it under an electron microscope; I am grateful to Robley Williams for showing me how to metal shadow my samples (Pemberton,1973; Pemberton and AJ Clark,1973; Miller, Pemberton and Richards,1974;Pemberton,1974;Miller,Pemberton and Clark,1977). After advice from John Clark and when I returned to Australia and took up an appointment with UQ I decided to diversify my research. During my postdoc I worked alongside Anne Emerick who was working with the CAM (camphor degradation) plasmid. John Clark put me on her advisory panel (alongside Mike Doudoroff and Norberto Palleroni) making her my first PhD student. The bacterial degradation of such complex naturally occurring molecules such as camphor required a large number of steps requiring a large number of genes hence a large plasmid. I decided to determine if soil bacteria had evolved plasmids which encoded the degradation of man-made molecules. I chose the synthetic herbicide 2,4-D. My research was the first to identify, isolate and clone genes responsible for the degradation of a man-made molecule –moreover the 2,4-D degradation was encoded by a broad host range plasmid, providing an explanation of how microorganisms rapidly evolve the ability to degrade and recycle a vast array of worldwide synthetic environmental pollutants which cause a range of diseases from cancer to birth defects (Pemberton & Fisher, Nature, 1977). One of the most widely studied microorganisms is the bacterium Ralstonia eutropha JMP134 pJP4 (Hgr) which has an extraordinary ability to degrade and recycle the most complex and most toxic synthetic molecules (Don and Pemberton, J.Bacteriol, 1981;Schmidt et.al.,2011. Catabolic Plasmids.Encyclopedia of Life Sciences). Famously more recent studies have shown that there are genes and gene clusters encoding the degradation of plastics, explosives and chemical weapons of war . Detailed studies of bacterial genes involved in the environmental degradation and recycling of a wide range naturally occurring and synthetic molecules show that degradation genes and degradation gene clusters play a major role in the worldwide carbon cycle.

Photosynthesis is considered the most important biological process on earth. And one of the most intensively studied photosynthetic organisms is the bacterium Rhodobacter sphaeroides. To start the research a local strain of R.sphaeroides, designated RS601, was isolated by Bill Tucker (my first australian PhD student) from a water sample obtained from a roadside ditch in Brisbane (Pemberton and Tucker,1977;Tucker and Pemberton,1978;1979;1980). One of the first discoveries made with this strain was lysogenic conversion to antibiotic resistance by a naturally occurring virus .(JM Pemberton, WT Tucker - Nature, 1977).

Subsequently when this strain was infected withe the broad host plasmid RP1 carrying the mecuric ion transposon Tn501 chromosome transfer occurred. This allowed the construction of the first genetic map of a photosynthetic bacterium(Pemberton and Bowen, J.Bacteriol, 1981). Mapping revealed that the photosynthesis gene cluster was on the main chromosome. Remarkably chromosome transfer occurred from a site right next to the photosynthesis gene cluster with early transfer of the entire cluster into the recipent cell. This provides a potential mechanism for the evolution and spread of photosynthesis genes. A clone bank of RS601 was constructed using pHC79:: Tn5deltaBamH1. This vector allowed cosmid cloning into the BamH1 site of Tn5. These Tn5 cosmid clones were transposed onto the broad host range plasmid pR751. The ability to transfer the entire cosmid clone bank to a wide range of bacteria led to the first cloning and heterologous expression of a carotenoid gene cluster (Pemberton&Harding,Current Microbiology,1986 & 1987).This indicated that genes involved in photosynthesis could be transferred to and expressed in a range of unrelated non-photosynthetic bacteria. Subsequent heterologous expression of carotenoid genes in an increasing variety of plants led to the production of foods enriched in the precursors of vitamin A e.g. Golden Rice (Erik Stokstad, Science Nov 20, 2019) . Vitamin A deficiency is the major preventable cause of blindness in children under 5 years of age; it affects up to 500,000 children each year. Using the same clone bank in mapping experiments in Rhodobacter sphaeroides I observed a few pale colonies in which carotenoid biosynthesis was suppressed. Subsequent detailed analysis of one of these cosmids led to the discovery of the long sought master regulator (PpsR) of bacterial photosynthesis and provided the first detailed insight into the mechanism by which bacterial photosynthesis is regulated at the molecular level (A Gene from the Photosynthetic Gene Cluster of Rhodobacter sphaeroides Induces trans Suppression of Bacteriochlorophyll and Carotenoid Levels in R.sphaeroides and R.capsulatus (R.J.Penfold and JM Pemberton, Current Microbiology, 1991; Sequencing, Chromosomal Inactivation and Functional Expression in E.coli of ppsR a Gene which represses carotenoid and bacteriochlorophyll synthesis in Rhodobacter sphaeroides. RJ Penfold and JM Pemberton. J.Bacteriol May 1994).Early studies by Cohen-Bazire, Sistrom and Stanier (1957) revealed that oxygen and blue light had varying effects on photosynthesis in Rhodobacter. The effect of oxygen was profound. The effect of blue light was more muted. The initial sequencing of ppsR (Penfold and Pemberton, 1994) revealed the presence of only two cys residues suggesting a possible mechanism for the profound effect of oxygen on PpsR repressor activity. Studies of conformational changes/repressor activity of PpsR in the presence and absence of oxygen have produced mixed results(Gomelsky et al.,2000;Masuda and Bauer.,2002). In contrast the muted effect of blue light on photosynthesis appears to be due to the blue light sensitive, anti-repressor AppA. (Gomelsky and Kaplan,1995). It is not known if any other environmental signals modulate PpsR activity.The rhodobacter research led to the construction of pJP5603 which allowed the precise insertion of a defined segment of DNA into a bacterial genome (Penfold and Pemberton,1992 ; Zordan,Beliveau,Trow,Craig and Cormack, 2015). The technique was used to either add functional genes or groups of genes to a precise location in the genome or to precisely target and inactivate individual genes. The site of insertion/mutagenesis is tagged with an antibiotic resistance gene. This process is known as “recombineering” ( Zhang et al., 1988). As with all forms of mutagenesis there are “off target” mutations. The consequences of such ”off target” mutations can range from minimal to extensive.

In a study of a range of genes encoding secreted enzymes involved in the degradation of naturally-occurring biological polymers e.g xylanases, cellulases,amylases, chitinases etc I attempted to obtain secretion genes from Chromobacterium violaceum. Again using the pHC79:: Tn5deltaBamH1 vector used in the study of the photosynthesis genes (Pemberton&Harding,Current Microbiology,1986 & 1987) I constructed a cosmid clone bank of C.violaceum. The clone bank I constructed did not produce secretion genes but instead 2-3 of the clones expressed the intense purple pigmented violacein in E.coli(Pemberton,1986). Subsequent subcloning revealed the gene cluster occupied 8kb and transposon mutagenesis revealed intense blue and intense green intermediates. (Pemberton et.al.,Current Microbiology,1991). I am grateful to Trudy Grossman for the detailed study of this cluster which included sequence analysis and functional characterisation of the violacein biosynthetic pathway (August et al., 2000). The functional analysis of the violacein gene cluster revealed that VioA VioC and VioD belong to the PheA(phenol) /TfdB (2,4-D) group of FAD dependant mono-oxygenases. TfdB is encoded by the 2,4-D degradation gene cluster of the broad host range IncP plasmid pJP4 carried by Ralstonia eutropha JMP134. This provides a link between the degradation of a man-made molecule-2,4-D and the synthesis of an anti-tumour antibiotic-violacein. Remarkably, under certain circumstances this 2,4-D degradation pathway can convert 2,4-D into the well known plant antibiotic-protoanemonin (Blasco,R et al., 1995).In 1983 Burt Ensley , Barry Ratzkin and co-workers (Ensley et al.,Science,1983) discovered that the naphthalene dioxygenase gene from Pseudomonas putida enabled E.coli K12 to synthesise the famous blue dye indigo from tryptophan; a second gene, VioD, from the violacein gene cluster also enabled E.coli K12 to produce indigo (Cheah et al.,Acta Crystallographica,1998). Further studies using the violacein gene cluster led to the development of techniques and vectors that should allow cloning and stable, high level expression of more antibiotic biosynthesis pathways in E.coli K12, particularly pathways from the prolific antibiotic producers the Streptomycetes providing novel antibiotics in the fight against antibiotic resistant pathogens (Sarovich and Pemberton,2007; Philip,Sarovich and Pemberton,2008 & 2009;Ahmetagic & Pemberton, 2010 & 2011;Ahmetagic, Philip ,Sarovich,Kluver and Pemberton,2011).An article published in June 2013 by Stevens and co-workers PLoS ONE 8(5) showed that a native gene cluster from Streptomyces rimosus encoding tetracycline can be directly expressed in E.coli K12.

For the first time researchers have showed the expression of the violacein gene cluster in a eukaryote-the yeast Saccharomyces cerevisiae (Lee et al., 2013). Such a discovery may indicate that the violacein gene cluster can be expressed in organisms which range from microbes to man. It may also indicate that major pathways from microorganisms can be engineered and expressed in a range of eukaryotes. Since violacein is a potent anticancer agent it is of interest to determine if the violacein cluster engineered into bacteria of the microbiome of an animal reduces cancer rates. Alternatively it may be possible to engineer the violacein pathway directly into an animal and observe if cancer rates are reduced. In view of the purported prokaryotic ancestry of eukaryoyic organelles such as mitochondria and chloroplasts ,one possible way of boosting violacein synthesis in eukaryotic cells could be to integrate the violacein gene cluster into organelle DNA.

Finally, violacein is chemically related to the well known anti-cancer drug staurosporine and possesses anticancer, antifungal, anti-parasite, antibacterial and antiviral activities;it might be possible to synthesise structural variants of violacein with more potent activity against various cancers and drug/antibiotic resistant pathogens. Interestingly is now known that violacein producing bacteria associated with the skin microbiome of certain frogs provides some protection against extinction by the worldwide spread of ‘chytrid’ fungus(Harris et.al., 2009). In addition, frogs have been used in cancer studies and may provide a simple model to test the anticancer properties of violacein. Since the violacein gene cluster is expressed in a wide range of bacteria ( Dr D S Philip, personal communication;D.S/Philip.PhD Thesis 2010) and has potent activity against the malarial parasite Plasmodium falciparum and other mosquito borne parasites, there is the possibility that mosquitoes engineered to carry the violacein gene cluster might be resistant to parasite infection. The cluster could be stably incorporated in the genomes of bacteria normally inhabiting the surface or the gut of the mosquito.A recent patent application (United States Patent Application 20170280730) indicates that Chromobacterium introduced into the microbiome of mosquitoes is useful for the prevention of transmission of malaria and dengue virus.In addition, chemical modification of violacein may produce drugs with even higher levels of activity against parasites including the malarial parasite ( Wilkinson et al., 2020). Violacein has activity against the pandemic virus Covid19 and there is some knowledge of its mode of action (Duran et al., 2021). Testing may reveal if it has activity against both Kappa and Delta Covid19 variants.Violacein can inhibit infection by HIV and COVID (Doganci et al., 2022)

• Fellow, American Society for Microbiology
• Fellow, Australian Society for Microbiology

Selected Publications:

• Ahmetagic, Adnan, Philip, Daniel S., Sarovich, Derek S., Kluver, Daniel W. and Pemberton, John M. (2011) Plasmid encoded antibiotics inhibit protozoan predation of Escherichia coli K12. Plasmid, 66 3: 152-158. doi:10.1016/j.plasmid.2011.07.006
• Ahmetagic, Adnan and Pemberton, John M. (2011) Antibiotic resistant mutants of Escherichia coli K12 show increases in heterologous gene expression. Plasmid, 65 1: 51-57. doi:10.1016/j.plasmid.2010.11.004
• Ahmetagic, Adnan and Pemberton, John M. (2010) Stable high level expression of the violacein indolocarbazole anti-tumour gene cluster and the Streptomyces lividans amyA gene in E. coli K12. Plasmid, 63 2: 79-85. doi:10.1016/j.plasmid.2009.11.004
• Philip, Daniel S., Sarovich, Derek S. and Pemberton, John M. (2009) Complete sequence and analysis of the stability functions of pPSX, a vector that allows stable cloning and expression of Streptomycete genes in Escherichia coli K12. Plasmid, 62 1: 39-43. doi:10.1016/j.plasmid.2009.03.002

Emeritus Professor Michael Pender graduated from The University of Queensland in 1974 with First Class Honours in Medicine and a University Medal. Over the next six years he trained as a physician and neurologist at the Royal Prince Alfred Hospital and St Vincent's Hospital, Sydney, and became a Fellow of the Royal Australasian College of Physicians (FRACP) in 1981. During his specialist clinical training he developed a keen interest in multiple sclerosis which he has continued since then. After completing his clinical training in neurology, he was a research scholar in the field of multiple sclerosis at the Institute of Neurology, Queen Square, London, and was awarded a PhD from the University of London and Queen Square Prize for Research in 1983. From 1984 to 1986 he continued this research as a Research Fellow at the John Curtin School of Medical Research, Australian National University, Canberra. In 1987 he was appointed Senior Lecturer in the Department of Medicine, The University of Queensland, at the Royal Brisbane Hospital. In 1989 he was awarded a Doctorate of Medicine from The University of Queensland for his research in the field of multiple sclerosis and was promoted to Reader in Medicine. In 1995 he was promoted to Professor of Medicine (Personal Chair), The University of Queensland, which he held until his retirement in 2021. His main clinical and research interest is multiple sclerosis. He also held the positions of: Consultant Neurologist, Royal Brisbane and Women's Hospital, 1987–2021; Director of Neurology, Royal Brisbane and Women's Hospital, 1992–2005: Director of the Neuroimmunology Research Centre, The University of Queensland, 1991–2007; Director of the Multiple Sclerosis Research Centre, The University of Queensland, 2009–2014; and Clinical Fellow, QIMR Berghofer Medical Research Institute, 2017–2021. In 1996, with the support of the Multiple Sclerosis Society of Queensland, he established a Multiple Sclerosis Clinic at the Royal Brisbane and Women's Hospital. In 2006 he was awarded the Multiple Sclerosis Australia Prize for Multiple Sclerosis Research - "For outstanding commitment and dedication to research into the cause and cure of Multiple Sclerosis in Australia". In 2011 he received the John H Tyrer Prize in Internal Medicine, The University of Queensland, for research in the field of Internal Medicine. He was the Sir Raphael Cilento Orator of the Royal Australasian College of Medical Administrators for 2009 and the W Ian McDonald Lecturer of the Australian and New Zealand Association of Neurologists for 2014. In 2019 he received the John Studdy Award from Multiple Sclerosis Australia for "lifelong commitment and service to research to identify the cause of and potential cure for Multiple Sclerosis". In 2024 he was appointed a Member of the Order of Australia for significant service to medicine, particularly neurology and multiple sclerosis research, and to tertiary education. Major research achievements include: the discovery of apoptosis of autoreactive T cells in the central nervous system as a fundamental mechanism of recovery from autoimmune attack (Journal of the Neurological Sciences 1991, Journal of Autoimmunity 1992, European Journal of Immunology 1994); formulation of a novel hypothesis (The Lancet 1998) proposing a failure of this mechanism in multiple sclerosis; and the further development of this hypothesis into a new paradigm (Trends in Immunology 2003) for the cause of human chronic autoimmune diseases based on infection of autoreactive B cells with the Epstein–Barr virus (EBV), some of the predictions of which have already been verified in multiple sclerosis, rheumatoid arthritis and Sjögren’s syndrome. His EBV hypothesis led to the first clinical trial of EBV-specific T cell therapy in multiple sclerosis (JCI Insight 2018), a trial in which he was a principal investigator.

Group page: https://medicine-program.uq.edu.au/multiple-sclerosis-research-group

Dr Natalie Peng is a lecturer in Accounting at the UQ Business School.

Natalie has taught both financial and management accounting courses at UQ.

Her research interests include superannuation, the production, dissemination and interpretation of information in the financial market, disclosure regulation, and cognitive bias in decision-making and its consequences.

Dr. Xiyue Peng is a Postdoctoral Research Fellow at the Australian Institute for Bioengineering and Nanotechnology (AIBN), The University of Queensland. In 2023, she got her PhD from AIBN at UQ under the supervision of A/Prof. Bin Luo and Prof.Lianzhou Wang.

Dr. Xiyue Peng specializes in electrochemical energy storage and conversion systems, focusing primarily on the functional materials and energy storage mechanisms for rechargeable aluminum ion batteries, lithium ion batteries and redox flow batteries.