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2008 Journal Article Receptor for advanced glycation end products (RAGE) deficiency attenuates the development of atherosclerosis in diabetesSoro-Paavonen, Aino, Watson, Anna M. D., Li, Jiaze, Paavonen, Karri, Koitka, Audrey, Calkin, Anna C., Barit, David, Coughlan, Melinda T., Drew, Brian G., Lancaster, Graeme I., Thomas, Merlin, Forbes, Josephine M., Nawroth, Peter P., Bierhaus, Angelika, Cooper, Mark E. and Jandeleit-Dahm, Karin A. (2008). Receptor for advanced glycation end products (RAGE) deficiency attenuates the development of atherosclerosis in diabetes. Diabetes, 57 (9), 2461-2469. doi: 10.2337/db07-1808 |
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2008 Journal Article Cardiac inflammation associated with a Western diet is mediated via activation of RAGE by AGEsTikellis, Christos, Thomas, Merlin C., Harcourt, Brooke E., Coughlan, Melinda T., Pete, Josepha, Bialkowski, Katarzyna, Tan, Adeline, Bierhaus, Angelika, Cooper, Mark E. and Forbes, Josephine M. (2008). Cardiac inflammation associated with a Western diet is mediated via activation of RAGE by AGEs. American Journal of Physiology: Endocrinology and Metabolism, 295 (2), E323-E330. doi: 10.1152/ajpendo.00024.2008 |
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2008 Conference Publication Urinary, but not serum levels of the AGE, n-carboxymethyl-lysine are associated with progressive nephropathy in patients with type 1 and type 2 diabetesCoughlan, Melinda T., Penfold, Sally A., Cooper, Mark E., Panagiotopoulos, Sianna, Macisaac, Richard J., Jerums, George and Forbes, Josephine M. (2008). Urinary, but not serum levels of the AGE, n-carboxymethyl-lysine are associated with progressive nephropathy in patients with type 1 and type 2 diabetes. 67th Annual Meeting of the American-Diabetes-Association, San Francicso Ca, Jun 06-10, 2008. AMER DIABETES ASSOC. |
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2008 Journal Article Response to comment on: Thallas-bonke et al. (2008) inhibition of NADPH oxidase prevents advanced glycation end product-mediated damage in diabetic nephropathy through a protein kinase C-α-dependent pathway:Diabetes 57:460-469, 2008Thallas-Bonke, Vicki and Forbes, Josephine M. (2008). Response to comment on: Thallas-bonke et al. (2008) inhibition of NADPH oxidase prevents advanced glycation end product-mediated damage in diabetic nephropathy through a protein kinase C-α-dependent pathway:Diabetes 57:460-469, 2008. Diabetes, 57 (6), e14-e14. doi: 10.2337/db08-0406 |
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2008 Conference Publication The renal pathogenicity of dietary glycotoxins is mediated via the receptor for advanced glycation end products (RAGE)Coughlan, Melinda T., Mibus, Amy L., Tan, Adeline L., Harcourt, Brooke E., Sourris, Karly C., Penfold, Sally A., Thallas-Bonke, Vicki, Yap, Felicia Y., Gasser, Anna, Thomas, Merlin, Su, Qing, Bierhaus, Angelika, Cooper, Mark E. and Forbes, Josephine M. (2008). The renal pathogenicity of dietary glycotoxins is mediated via the receptor for advanced glycation end products (RAGE). 67th Annual Meeting of the American-Diabetes-Association, San Francicso Ca, Jun 06-10, 2008. AMER DIABETES ASSOC. |
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2008 Journal Article Oxidative stress as a major culprit in kidney disease in diabetesForbes, Josephine M., Coughlan, Melinda T. and Cooper, Mark E. (2008). Oxidative stress as a major culprit in kidney disease in diabetes. Diabetes, 57 (6), 1446-1454. doi: 10.2337/db08-0057 |
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2008 Journal Article MKK3 signalling plays an essential role in leukocyte-mediated pancreatic injury in the multiple low-dose streptozotocin modelFukuda, Kyoichi, Tesch, Greg H., Yap, Felicia Y., Forbes, Josephine M., Flavell, Richard A., Davis, Roger J. and Nikolic-Paterson, David J. (2008). MKK3 signalling plays an essential role in leukocyte-mediated pancreatic injury in the multiple low-dose streptozotocin model. Laboratory Investigation, 88 (4), 398-407. doi: 10.1038/labinvest.2008.10 |
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2008 Journal Article Inhibition of NADPH oxidase prevents advanced glycation end product-mediated damage in diabetic nephropathy through a protein kinase C-α-dependent pathwayThallas-Bonke, Vicki, Thorpe, Suzanne R., Coughlan, Melinda T., Fukami, Kei, Yap, Felicia Y. T., Sourris, Karly C., Penfold, Sally A., Bach, Leon A., Cooper, Mark E. and Forbes, Josephine M. (2008). Inhibition of NADPH oxidase prevents advanced glycation end product-mediated damage in diabetic nephropathy through a protein kinase C-α-dependent pathway. Diabetes, 57 (2), 460-469. doi: 10.2337/db07-1119 |
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2008 Conference Publication Oxidative stress and advanced glycation in diabetic nephropathyCoughlan, Melinda T., Mibus, Amy L. and Forbes, Josephine M. (2008). Oxidative stress and advanced glycation in diabetic nephropathy. The 9th International Symposium on the Maillard Reaction, Munich, Germany, 1-5 September 2007. Malden, MA, United States: Wiley-Blackwell Publishing. doi: 10.1196/annals.1433.018 |
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2008 Journal Article Advanced glycation: implications in tissue damage and diseaseGasser, Anna and Forbes, Josephine M. (2008). Advanced glycation: implications in tissue damage and disease. Protein and Peptide Letters, 15 (4), 385-391. doi: 10.2174/092986608784246515 |
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2008 Conference Publication Therapeutic interruption of advanced glycation in diabetic nephropathy: do all roads lead to Rome?Sourris, Karly C., Forbes, Josephine M. and Cooper, Mark E. (2008). Therapeutic interruption of advanced glycation in diabetic nephropathy: do all roads lead to Rome?. The 9th International Symposium on the Maillard Reaction, Munich, Germany, 1-5 September 2007. Malden, MA, United States: Wiley-Blackwell Publishing. doi: 10.1196/annals.1433.048 |
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2007 Conference Publication Interaction between the receptor for advanced glycation end products (RAGE) and estrone (E1) in the development of type 2 diabetic nephropathyHarcourt, B. E., Mibus, A., Gasser, A., Andikopoulos, S., Thomas, M., Coughlan, M. T., Sourris, K., Rice, G., Bierhaus, A., Cooper, M. E. and Forbes, J. M. (2007). Interaction between the receptor for advanced glycation end products (RAGE) and estrone (E1) in the development of type 2 diabetic nephropathy. 43rd Annual Meeting of the European-Association-for-the-Study-of-Diabetes, Amsterdam Netherlands, Sep 18-21, 2007. SPRINGER. |
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2007 Journal Article Advanced glycation of apolipoprotein A-I impairs its anti-atherogenic propertiesHoang, A., Murphy, A. J., Coughlan, M. T., Thomas, M. C., Forbes, J. M., O'Brien, R., Cooper, M. E., Chin-Dusting, J. P. F. and Sviridov, D. (2007). Advanced glycation of apolipoprotein A-I impairs its anti-atherogenic properties. Diabetologia, 50 (8), 1770-1779. doi: 10.1007/s00125-007-0718-9 |
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2007 Journal Article Combination therapy with the advanced glycation end product cross-link breaker, alagebrium, and angiotensin converting enzyme inhibitors in diabetes: synergy or redundancy?Coughlan, Melinda T., Thallas-Bonke, Vicki, Pete, Josefa, Long, David M., Gasser, Anna, Tong, David C. K., Arnstein, Maryann, Thorpe, Suzanne R., Cooper, Mark E. and Forbes, Josephine M. (2007). Combination therapy with the advanced glycation end product cross-link breaker, alagebrium, and angiotensin converting enzyme inhibitors in diabetes: synergy or redundancy?. Endocrinology, 148 (2), 886-895. doi: 10.1210/en.2006-1300 |
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2007 Journal Article AGE, RAGE, and ROS in diabetic nephropathyTon, Adeline L. Y., Forbes, Josephine M. and Cooper, Mark E. (2007). AGE, RAGE, and ROS in diabetic nephropathy. Seminars in Nephrology, 27 (2), 130-143. doi: 10.1016/j.semnephrol.2007.01.006 |
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2007 Journal Article Renal microvascular injury in diabetes: RAGE and redox signalingCoughlan, Melinda T., Cooper, Mark E. and Forbes, Josephine M. (2007). Renal microvascular injury in diabetes: RAGE and redox signaling. Antioxidants and Redox Signaling, 9 (3), 331-342. doi: 10.1089/ars.2006.1469 |
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2007 Conference Publication Role of the AGE crosslink breaker, alagebrium, as a renoprotective agent in diabetesCoughlan, M. T., Forbes, J. M. and Cooper, M. E. (2007). Role of the AGE crosslink breaker, alagebrium, as a renoprotective agent in diabetes. 5th Hyonam-Kidney-Laboratory-International-Diabetes Symposium, Seoul South Korea, Feb 02-03, 2007. Nature Publishing Group. doi: 10.1038/sj.ki.5002387 |
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2007 Journal Article Diabetic nephropathy: where hemodynamics meets metabolismForbes, J. M., Fukami, K. and Cooper, M. E. (2007). Diabetic nephropathy: where hemodynamics meets metabolism. Experimental and Clinical Endocrinology and Diabetes, 115 (2), 69-84. doi: 10.1055/s-2007-949721 |
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2006 Conference Publication Diabetes-induced effects on endothelial function and the actions of nitric oxideSoro-Paavonen, A., Zhang, W. -Z., Venardos, K., Jones, E., Chin-Dusting, J., Thomas, M. T., Coughlan, M. T., Brasacchio, D., Cooper, M. E., Kaye, D. and Forbes, J. M. (2006). Diabetes-induced effects on endothelial function and the actions of nitric oxide. 42nd Annual Meeting of the European-Association-for-the-Study-of-Diabetes (EASD), Copenhagen Denmark, Sep 14-17, 2006. SPRINGER. |
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2006 Journal Article Connective tissue growth factor plays an important role in advanced glycation end product-induced tubular epithelial-to-mesenchymal transition: implications for diabetic renal diseaseBurns, Wendy C., Twigg, Stephen M., Forbes, Josephine M., Pete, Josefa, Tikellis, Christos, Thallas-Bonke, Vicki, Thomas, Merlin C., Cooper, Mark E. and Kantharidis, Phillip (2006). Connective tissue growth factor plays an important role in advanced glycation end product-induced tubular epithelial-to-mesenchymal transition: implications for diabetic renal disease. Journal of the American Society of Nephrology, 17 (9), 2484-2494. doi: 10.1681/ASN.2006050525 |
