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2024

Conference Publication

Endothelial Protein C Receptor CD201 Enables Identification of Transplantable Mouse Hematopoietic Stem Cells in Inflammatory Conditions Unlike SCA1

Bisht, Kavita, Shatunova, Svetlana, Barbier, Valerie, Winkler, Ingrid G. and Levesque, Jean-Pierre (2024). Endothelial Protein C Receptor CD201 Enables Identification of Transplantable Mouse Hematopoietic Stem Cells in Inflammatory Conditions Unlike SCA1. 66th ASH Annual Meeting, San Diego, CA United States, 7-10 December 2024. Washington, DC United States: American Society of Hematology. doi: 10.1182/blood-2024-199946

Endothelial Protein C Receptor CD201 Enables Identification of Transplantable Mouse Hematopoietic Stem Cells in Inflammatory Conditions Unlike SCA1

2024

Conference Publication

Endotoxemia As Possible Cause of Inflammatory Bowel Diseases-Associated Anemia

Bisht, Kavita, An, Yoon-Kyo, Shatunova, Svetlana, Wang, Ran, Barbier, Valerie, Giri, Rabina, Amiss, Anna, Tang, Yifu, Alexander, Kylie, Millard, Susan, Winkler, Ingrid G., Pettit, Allison, Begun, Jakob and Levesque, Jean-Pierre (2024). Endotoxemia As Possible Cause of Inflammatory Bowel Diseases-Associated Anemia. 66th ASH Annual Meeting, San Diego, CA United States, 7-10 December 2024. Washington, DC United States: American Society of Hematology. doi: 10.1182/blood-2024-193349

Endotoxemia As Possible Cause of Inflammatory Bowel Diseases-Associated Anemia

2024

Conference Publication

The Endothelial Protein C Receptor CD201 Identifies Transplantable Mouse Hematopoietic Stem Cells In Inflammatory Conditions Unlike SCA-1

Bisht, Kavita, Shatunova, Svetlana, Barbier, Valerie, Winkler, Ingrid and Levesque, Jean-Pierre (2024). The Endothelial Protein C Receptor CD201 Identifies Transplantable Mouse Hematopoietic Stem Cells In Inflammatory Conditions Unlike SCA-1. 53rd Annual Scientific Meeting ISEH 2024, Chicago, IL United States, 29 August - 1 September 2024. Philadelphia, PA United States: Elsevier. doi: 10.1016/j.exphem.2024.104348

The Endothelial Protein C Receptor CD201 Identifies Transplantable Mouse Hematopoietic Stem Cells In Inflammatory Conditions Unlike SCA-1

2019

Conference Publication

Erythropoiesis suppression by bacterial liposaccharides is extrinsically mediated independently of G-CSF

Bisht, Kavita , Tay, Joshua , Jacobsen, Rebecca , McGirr, Crystal , Fleming, Whitney , Nowlan, Bianca , Barbier, Valerie , Winkler, Ingrid and Levesque, Jean-Pierre (2019). Erythropoiesis suppression by bacterial liposaccharides is extrinsically mediated independently of G-CSF. International Society of Experimental Hematology, Brisbane, Australia, 22-25 August 2019. Philadelphia, PA, United States: Elsevier.

Erythropoiesis suppression by bacterial liposaccharides is extrinsically mediated independently of G-CSF

2016

Conference Publication

Suppression of medullar erythropoiesis in response to bacterial lipopolysaccharides (LPS) involves two distinct TLR4-dependent mechanisms with contrasted requirements for G-CSF receptors

Bisht, Kavita, Jacobsen, Rebecca, Nowlan, Bianca, McGirr, Crystal, Brunck, Marion, Keech, Thomas, Winkler, Ingrid G. and Levesque, Jean-Pierre (2016). Suppression of medullar erythropoiesis in response to bacterial lipopolysaccharides (LPS) involves two distinct TLR4-dependent mechanisms with contrasted requirements for G-CSF receptors. 58th Annual Meeting and Exposition of the American Society of Hematology (ASH), San Diego, CA, United States, 3-6 December 2016. Washington, DC, United States: American Society of Hematology. doi: 10.1182/blood.V128.22.546.546

Suppression of medullar erythropoiesis in response to bacterial lipopolysaccharides (LPS) involves two distinct TLR4-dependent mechanisms with contrasted requirements for G-CSF receptors

2016

Conference Publication

Hematopoietic Stem Cell Mobilization and Erythropoiesis Suppression in Response to Lipopolysaccharides Involve Two Distinct Tlr4-Depedent Mechanisms with Different Requirement for G-Csf Receptors

Bisht, Kavita, Jacobsen, Rebecca, Brunck, Marion, Keech, Thomas, McGirr, Crystal, Nowlan, Bianca, Barbier, Valerie, Winkler, Ingrid and Levesque, Jean-Pierre (2016). Hematopoietic Stem Cell Mobilization and Erythropoiesis Suppression in Response to Lipopolysaccharides Involve Two Distinct Tlr4-Depedent Mechanisms with Different Requirement for G-Csf Receptors. 45th Annual Scientific Meeting of the International-Society-for-Experimental-Hematology (ISEH), San Diego, CA, United States, 25-28 August 2016. Philadelphia, PA, United States: Elsevier. doi: 10.1016/j.exphem.2016.06.096

Hematopoietic Stem Cell Mobilization and Erythropoiesis Suppression in Response to Lipopolysaccharides Involve Two Distinct Tlr4-Depedent Mechanisms with Different Requirement for G-Csf Receptors

2015

Conference Publication

Carbon monoxide targets Notch1 and MAPK-ERK1/2 signaling pathways to block growth of lung carcinoma

Nemeth, Zsuzsanna, Csizmadia, Eva, Vikstrom, Lisa, Li, Mailin, Bisht, Kavita, Feizi, Alborz, Gallo, David, Otterbein, Leo, Fillinger, Janos, Dome, Balazs, B. Costa, Daniel and Wegiel, Barbara (2015). Carbon monoxide targets Notch1 and MAPK-ERK1/2 signaling pathways to block growth of lung carcinoma. AACR 106th Annual Meeting 2015, Philadelphia, PA, United States, 18-22 April 2015. Philadelphia, PA, United States: American Association for Cancer Research. doi: 10.1158/1538-7445.am2015-416

Carbon monoxide targets Notch1 and MAPK-ERK1/2 signaling pathways to block growth of lung carcinoma

2012

Conference Publication

Reduced LDL oxidation is secondary to protection from in vivo thiol oxidation and hypocholesterolemia in Gilbert's syndrome

Bulmer, Andrew Cameron, Boon, Ai-Ching, Hawkins, Clare, Bisht, Kavita, Coombes, Jeff and Wagner, Karl-Heinz (2012). Reduced LDL oxidation is secondary to protection from in vivo thiol oxidation and hypocholesterolemia in Gilbert's syndrome. Experimental Biology Meeting, San Diego, United States, 21-25 April 2012. Bethesda, MD, United States: Federation of American Societies for Experimental Biology.

Reduced LDL oxidation is secondary to protection from in vivo thiol oxidation and hypocholesterolemia in Gilbert's syndrome