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Store operated Ca2+ entry in skeletal fibres from normal and dystrophin-deficient muscle (2007-2010)

Abstract

Skeletal muscle, the most abundant tissue in the body, is essential for life and movement. Muscle function is regulated in a complex manner by calcium and is severely impaired in patients with Duchenne muscular dystrophy. Changes in calcium regulation, known to occue in dystrophic muscle, will be investigated in an animal model using a novel, single cell approach. Results obtained will contribute to understanding better muscular dystrophy and help provide therapeutic targets for treatment.

Experts

Professor Bradley Launikonis

Affiliate of Centre for Research on Exercise, Physical Activity and Health
Centre for Research on Exercise, Physical Activity and Health
Faculty of Health, Medicine and Behavioural Sciences
Professor
School of Biomedical Sciences
Faculty of Health, Medicine and Behavioural Sciences
Bradley Launikonis