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2024 Conference Publication Endotoxemia As Possible Cause of Inflammatory Bowel Diseases-Associated AnemiaBisht, Kavita, An, Yoon-Kyo, Shatunova, Svetlana, Wang, Ran, Barbier, Valerie, Giri, Rabina, Amiss, Anna, Tang, Yifu, Alexander, Kylie, Millard, Susan, Winkler, Ingrid G., Pettit, Allison, Begun, Jakob and Levesque, Jean-Pierre (2024). Endotoxemia As Possible Cause of Inflammatory Bowel Diseases-Associated Anemia. 66th ASH Annual Meeting, San Diego, CA United States, 7-10 December 2024. Washington, DC United States: American Society of Hematology. doi: 10.1182/blood-2024-193349 |
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2024 Conference Publication Endogenous glucocorticoids promote neurogenic heterotopic ossification after spinal cord injury and glucocorticoid receptor antagonist treatment prevents their developmentAlexander, Kylie, Tseng, Hsu-Wen, Girard, Dorothee, Barbier, Valerie, Lao, Hong Wa, Samuel, Selwin G., Ungerer, Jacobus P. J., McWhinney, Brett C., Fleming, Whitney, Salga, Marjorie, Genet, Francois, Ruitenberg, Mark J., Banzet, Sebastien and Levesque, Jean-Pierre (2024). Endogenous glucocorticoids promote neurogenic heterotopic ossification after spinal cord injury and glucocorticoid receptor antagonist treatment prevents their development. 2024 Annual Meeting of the American Society for Bone and Mineral Research (ASBMR), Toronto, ON, Canada, 27-30 September 2024. Cary, NC, United States: Oxford University Press. doi: 10.1093/jbmr/zjae183 |
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2018 Conference Publication Oncostatin M is a key effector of heterotopic ossification following spinal cord injuriesTseng, Hsu-Wen, Alexander, Kylie, Kulina, Irina, Salga, Marjorie, Jose, Beulah, Genet, Francois, Torossian, Frederic, Guerton, Bernadette, Anginot, Adrienne, Fleming, Whitney, Millard, Susan, Pettit, Allison, Sims, Natalie, Lataillade, Jean-Jacques, Le Bousse-Kerdiles, Marie-Caroline and Levesque, Jean Pierre (2018). Oncostatin M is a key effector of heterotopic ossification following spinal cord injuries. Annual Meeting of the American Society for Bone and Mineral Research, Montreal Canada, September 28 - October 1 2018. Hoboken, NJ United States: Wiley. |
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2016 Conference Publication Autophagy-dependent TIGIT+ Treg are critical for the maintenance of toleranceMacDonald, Kelli P. A., Le Texier, Laetitia, Leveque-ElMouttie, Lucie, Lineburg, Katie, Nicholls, Jemma, Guimaraes, Fernando Souza-Fonseca, Alexander, Kylie, Clouston, Andrew, Blazar, Bruce R. and Hill, Geoff R. (2016). Autophagy-dependent TIGIT+ Treg are critical for the maintenance of tolerance. Immunology 2016 Meeting, Seattle, WA United States, 13-17 May 2016. Rockville, MD United States: American Association of Immunologists. doi: 10.4049/jimmunol.196.supp.125.14 |
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2015 Conference Publication Autophagy-Dependent Tigit Positive Helios Positive Regulatory T Cells Are Critical for Controlling Gvhd After Allogeneic Stem Cell TransplantationLe Texier, Laetitia, Leveque-ElMouttie, Lucie, Lineburg, Katie E., Alexander, Kylie A., Melino, Michelle, Teal, Bianca, Martinez, Michelle, Kuns, Rachel D., Lane, Steven W., Engwerda, Christian, Blazar, Bruce R., Hill, Geoffrey R. and MacDonald, Kelli P. A. (2015). Autophagy-Dependent Tigit Positive Helios Positive Regulatory T Cells Are Critical for Controlling Gvhd After Allogeneic Stem Cell Transplantation. 14th Transplantation Science Symposium of the Transplantation-Society (TSS), Lorne, VIC, Australia, 11-13 November 2015. Philadelphia, PA, United States: Lippincott Williams & Wilkins. |
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2014 Conference Publication Lung parenchyma-derived IL-6 promotes IL-17A-dependent acute lung injury after allogeneic stem cell transplantationVarelias, Antiopi, Gartlan, Kate H., Kreijveld, Ellen, Olver, Stuart D., Lor, Mary, Kuns, Rachel D., Lineburg, Katie E., Teal, Bianca E., Raffelt, Neil C., Cheong, Melody, Alexander, Kylie A., Koyama, Motoko, Markey, Kate A., Sturgeon, Elise, Leach, Justine, Reddy, Pavan, Kennedy, Glen, Yanik, Gregory, Blazar, Bruce R., Tey, Siok K., Clouston, Andrew, MacDonald, Kelli P. A., Cooke, Kenneth R. and Hill, Geoffrey R. (2014). Lung parenchyma-derived IL-6 promotes IL-17A-dependent acute lung injury after allogeneic stem cell transplantation. 2nd Annual Meeting of the International-Cytokine-and-Interferon-Society (ICIS), Melbourne, Australia, 26-29 October 2014. London, United Kingdom: Academic Press. doi: 10.1016/j.cyto.2014.07.195 |
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2014 Conference Publication CSF-1-dependant M2 macrophages mediate chronic graft-versus-host diseaseAlexander, K., Flynn, R., Lineburg, K., Kuns, R., Janela, B., Teal, B., Olver, S., Lor, M., Raffelt, N., Koyama, M., Leveque, L., Le Texier, L., Melino, M., Markey, K., Varelias, A., Ginhoux, F., Engwerda, C., Clouston, A., Blazer, B., Hill, G. and MacDonald, K. (2014). CSF-1-dependant M2 macrophages mediate chronic graft-versus-host disease. World Transplant Congress, San Francisco, CA, United States, 26-31 July 2014. Hoboken, NJ, United States: Wiley-Blackwell. |
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2014 Conference Publication CSF-1-Dependant M2 Macrophages Mediate Chronic Graft-Versus-Host DiseaseAlexander, K., Flynn, R., Lineburg, K., Kuns, R., Janela, B., Teal, B., Olver, S., Lor, M., Raffelt, N., Koyama, M., Leveque, L., Le Texier, L., Melino, M., Markey, K., Varelias, A., Ginhoux, F., Engwerda, C., Clouston, A., Blazer, B., Hill, G. and MacDonald, K. (2014). CSF-1-Dependant M2 Macrophages Mediate Chronic Graft-Versus-Host Disease. World Transplant Congress, San Francisco United States, Jul 26-31, 2014. Philadelphia United States: Lippincott Williams & Wilkins. doi: 10.1097/01.tp.0000452130.27963.71 |
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2013 Conference Publication Fracture Healing Via Periosteal Callus Formation Requires Macrophages for Both Initiation and Progression of Endochondral OssificationPettit, A., Raggatt, L., Wullschleger, M., Alexander, K., Steck, R., Kaur, S. and Wu, A. (2013). Fracture Healing Via Periosteal Callus Formation Requires Macrophages for Both Initiation and Progression of Endochondral Ossification. Annual Meeting of the American Society for Bone and Mineral Research, Baltimore MD United States, 4-7 October 2013. Hoboken, NJ United States: Wiley-Blackwell. |
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2013 Conference Publication Lung Parenchyma-Derived IL-6 Induces Alloantigen Specific Th17 Differentiation Within The Lung and Idiopathic Pneumonia Syndrome After Allogeneic Stem Cell TransplantationVarelias, Antiopi, Gartlan, Kate H., Olver, Stuart D., Lineburg, Katie E., Kuns, Rachel D., Lor, Mary, Teal, Bianca E., Cheong, Melody, Koyama, Motoko, Markey, Kate A., Alexander, Kylie, Budelsky, Alison, Sturgeon, Elise, Leach, Justine, Avery, Judy, Kennedy, Glen A., Siok-Tey, Clouston, Andrew D., MacDonald, Kelli P. A. and Hill, Geoff R. (2013). Lung Parenchyma-Derived IL-6 Induces Alloantigen Specific Th17 Differentiation Within The Lung and Idiopathic Pneumonia Syndrome After Allogeneic Stem Cell Transplantation. 55th Annual Meeting of the American Society of Hematology, New Orleans, LA United States, 7-10 December 2013. Washington, DC United States: American Society of Hematology. |
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2012 Conference Publication Promoting Regulation Via the Inhibition of DNAM-1 After TransplantationKoyama, Motoko, Kuns, Rachel D., Olver, Stuart D., Lineburg, Katie E., Lor, Mary, Teal, Bianca E., Raffelt, Neil C., Leveque, Lucie, Chan, Christopher J., Robb, Renee J., Markey, Kate A., Alexander, Kylie A., Varelias, Antiopi, Clouston, Andrew D., MacDonald, Kelli P. A., Smyth, Mark J. and Hill, Geoffrey R. (2012). Promoting Regulation Via the Inhibition of DNAM-1 After Transplantation. 54th Annual Meeting and Exposition of the American-Society-of-Hematology (ASH), Atlanta Ga, Dec 08-11, 2012. WASHINGTON: AMER SOC HEMATOLOGY. |
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2011 Conference Publication B Cells Do Not Influence Intramembranous Bone Modelling in VivoPettit, A. R., Kaur, S., Alexander, K. A., MacDonald, K. P. A. and Raggatt, L. J. (2011). B Cells Do Not Influence Intramembranous Bone Modelling in Vivo. IOF Regionals 2nd Asia-Pacific Osteoporosis and Bone Meeting / ANZBMS Annual Scientific Meeting held with the JSBMR, Gold Coast, QLD Australia, 4-8 September 2011. London, United Kingdom: Springer. |
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2009 Conference Publication Osteomacs maintain the endosteal hematopoietic stem cell niche and participate in mobilizationPettit, A. R., Sims, N. A., Winkler, I. G., Alexander, K. A., Helwani, F., Raggatt, L. J. and Levesque, J. P. (2009). Osteomacs maintain the endosteal hematopoietic stem cell niche and participate in mobilization. 2nd Joint Meeting of the International Bone and Mineral Society/Australian New Zealand Bone and Mineral Society, Sydney, Australia, 21-25 March, 2009. United States: Elsevier Inc.. doi: 10.1016/j.bone.2009.01.082 |
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2009 Conference Publication Osteomacs: osteoclast precursors during inflammatory bone disease but regulators of physiologic bone remodellingRaggatt, L. J., Chang, M. K., Alexander, K. A., Maylin, E. R., Walsh, N. C., Gravallese, E. M., Hume, D. A. and Pettit, A. R. (2009). Osteomacs: osteoclast precursors during inflammatory bone disease but regulators of physiologic bone remodelling. 2nd Joint Meeting of the International Bone & Mineral Society and the Australian & New Zealand Bone & Mineral Society, Sydney, Australia, 21-25 March, 2009. New York: Elsevier Science. doi: 10.1016/j.bone.2009.01.300 |
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2009 Conference Publication Osteomacs are Critical for Optimal Intramembranous Bone Formation in a Tibial Defect Model of Bone HealingAlexander, K. A., Raggatt, L., Chang, M., Maylin, E., Muller, R., Kohler, T., Wu, A., Hume, D. and Pettit, A. (2009). Osteomacs are Critical for Optimal Intramembranous Bone Formation in a Tibial Defect Model of Bone Healing. Australian Society of Bone and Mineral Research (ASBMR) 31st Annual Meeting, Denver, Colorado, USA, 11-15 September, 2009. United States: American Society for Bone and Mineral Research. |
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2009 Conference Publication Osteomacs are critical for optimal intramembranous bone formation in a tibial defect model of bone healingAlexander, K. A., Raggatt, L. J., Chang, M. K., Maylin, E. R., Muller, R., Kohler, T., Wu, A. C. K., Hume, D. A. and Pettit, A. R. (2009). Osteomacs are critical for optimal intramembranous bone formation in a tibial defect model of bone healing. 2nd Joint Meeting of the International Bone & Mineral Society and the Australian & New Zealand Bone & Mineral Society, Sydney, NSW, Australia, 21-25 March 2009. United States: Elsevier. doi: 10.1016/j.bone.2009.01.076 |
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2009 Conference Publication Osteomacs: Osteoclast Precursors During Inflammatory Bone Disease but Regulators of Physiologic Bone RemodelingRaggatt, L., Chang, M., Alexander, K., Maylin, E., Walsh, N., Gravallese, E., Hume, D. and Pettit, A. (2009). Osteomacs: Osteoclast Precursors During Inflammatory Bone Disease but Regulators of Physiologic Bone Remodeling. Australian Society of Bone and Mineral Research (ASBMR) 31st Annual Meeting, Denver, CO, United States, 11-15 September, 2009. United States: American Society for Bone and Mineral Research. |
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2008 Conference Publication Primary murine osteoblast cultures contain macrophages that enhance osteoblast mineralizationAlexander, K. A., Chang, M. K., Hume, D. A., Pettit, A. R., Raggatt, L., Ripoli, V. M. and Schroder, K. (2008). Primary murine osteoblast cultures contain macrophages that enhance osteoblast mineralization. 35th European Symposium on Calcified Tissues, Barcelona, Spain, 24-28 May 2008. New York, USA: Springer. doi: 10.1007/s00223-008-9118-5 |
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2008 Conference Publication Primary murine osteoblast cultures contain macrophages that enhance osteoblast mineralisationChang, M. K., Pettit, A. R., Schroder, K., Ripoll, V. M., Alexander, K. A., Hume, D. A. and Raggatt, L. (2008). Primary murine osteoblast cultures contain macrophages that enhance osteoblast mineralisation. ECTS 35th European Symposium on Calcified Tissues, Barcelona, Spain, 24-28 May 2008. New York, NY, U.S.A.: Springer New York. doi: 10.1007/s00223-008-9118-5 |
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2007 Conference Publication Osteal Macrophages: Novel regulators of Bone FormationRaggatt, L. J., Chang, M. K., Alexander, K. A., Kuiliwaba, J. S., Fazzalari, N. L., Hume, D. A. and Pettit, A. R. (2007). Osteal Macrophages: Novel regulators of Bone Formation. 29th Annual Meeting of the American Society for Bone and Mineral Research, Honolulu, HI, 16th -19th September, 2007. Washington, D.C., United States: American Society for Bone and Mineral Research. |
