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2005 Journal Article The Angiotensin II Type 2 Receptor Causes Constitutive Growth of Cardiomyocytes and Does Not Antagonize Angiotensin II Type 1 Receptor–Mediated HypertrophyD’Amore, Angelo, Black, M. Jane and Thomas, Walter G. (2005). The Angiotensin II Type 2 Receptor Causes Constitutive Growth of Cardiomyocytes and Does Not Antagonize Angiotensin II Type 1 Receptor–Mediated Hypertrophy. Hypertension, 46 (6), 1347-1354. doi: 10.1161/01.HYP.0000193504.51489.cf |
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2005 Journal Article Effect of intrauterine growth restriction on the number of cardiomyocytes in rat heartsCorstius, Hugo Brandt, Zimanyi, Monika A., Maka, Noori, Herath, Thiloshini, Thomas, Walter, Van Der Laarse, Arnoud, Wreford, Nigel G. and Black, M. Jane (2005). Effect of intrauterine growth restriction on the number of cardiomyocytes in rat hearts. Pediatric Research, 57 (6), 796-800. doi: 10.1203/01.PDR.0000157726.65492.CD |
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2005 Journal Article G Protein Coupling and Second Messenger Generation Are Indispensable for Metalloprotease-dependent, Heparin-binding Epidermal Growth Factor Shedding Through Angiotensin II Type-1 ReceptorMifune, M., Ohtsu, H., Suzuki, H., Nakashima, H., Brailoiu, E., Dun, N. J., Frank, G. D., Inagami, T., Higashiyama, S., Thomas, W. G., Eckhart, A. D., Dempsey, P. J. and Eguchi, S. (2005). G Protein Coupling and Second Messenger Generation Are Indispensable for Metalloprotease-dependent, Heparin-binding Epidermal Growth Factor Shedding Through Angiotensin II Type-1 Receptor. Journal of Biological Chemistry, 280 (28), 26592-26599. doi: 10.1074/jbc.M502906200 |
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2005 Journal Article Double Trouble for Type 1 Angiotensin Receptors in AtherosclerosisThomas, Walter G (2005). Double Trouble for Type 1 Angiotensin Receptors in Atherosclerosis. New England Journal of Medicine, 352 (5), 506-508. doi: 10.1056/NEJMcibr043928 |
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2005 Journal Article Helix I of β-Arrestin Is Involved in Postendocytic Trafficking but Is Not Required for Membrane Translocation, Receptor Binding, and InternalizationDinh, Diem T., Qian, Hongwei, Seeber, Ruth, Lim, Esther, Pfleger, Kevin, Eidne, Karin A. and Thomas, Walter G. (2005). Helix I of β-Arrestin Is Involved in Postendocytic Trafficking but Is Not Required for Membrane Translocation, Receptor Binding, and Internalization. Molecular Pharmacology, 67 (2), 375-382. doi: 10.1124/mol.104.004721 |
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2005 Journal Article Evaluation of the membrane-binding properties of the proximal region of the angiotensin II receptor (AT1A) carboxyl terminus by surface plasmon resonanceKamimori, Hiroshi, Unabia, Sharon, Thomas, Walter G. and Aguilar, Marie-Isabel (2005). Evaluation of the membrane-binding properties of the proximal region of the angiotensin II receptor (AT1A) carboxyl terminus by surface plasmon resonance. Analytical Sciences, 21 (2), 171-174. doi: 10.2116/analsci.21.171 |
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2005 Journal Article Dual Pathways for Nuclear Factor kB Activation by Angiotensin II in Vascular Smooth Muscle Phosphorylation of p65 by IkB Kinase and Ribosomal KinaseZhang, Liping, Cheng, Jizhong, Ma, Yewei, Thomas, Walter, Zhang, Jiqiang and Du, Jie (2005). Dual Pathways for Nuclear Factor kB Activation by Angiotensin II in Vascular Smooth Muscle Phosphorylation of p65 by IkB Kinase and Ribosomal Kinase. Circulation Research, 97 (10), 975-982. doi: 10.1161/01.RES.0000190589.52286.41 |
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2004 Journal Article Hijacking epidermal growth factor receptors by angiotensin II: New possibilities for understanding and treating cardiac hypertrophySmith N. J., Chan, H.-W., Osborne, J. E., Thomas, W. G. and Hannan, R. D. (2004). Hijacking epidermal growth factor receptors by angiotensin II: New possibilities for understanding and treating cardiac hypertrophy. Cellular and Molecular Life Sciences, 61 (21), 2695-2703. doi: 10.1007/s00018-004-4244-3 |
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2004 Journal Article p38 mitogen-activated protein kinase inhibition improves cardiac function and attenuates left ventricular remodeling following myocardial infarction in the ratSee, Fiona, Thomas, Walter, Way, Kerrie, Tzanidis, Alex, Kompa, Andrew, Lewis, Dion, Itescu, Silviu and Krum, Henry (2004). p38 mitogen-activated protein kinase inhibition improves cardiac function and attenuates left ventricular remodeling following myocardial infarction in the rat. Journal of the American College of Cardiology, 44 (8), 1679-1689. doi: 10.1016/j.jacc.2004.07.038 |
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2004 Journal Article Cardiovascular Role of Urotensin II: Effect of Chronic Infusion in the RatKompa, A. R., Thomas, W. G., See, F., Tzanidis, A., Hannan, R. D. and Krum, H. (2004). Cardiovascular Role of Urotensin II: Effect of Chronic Infusion in the Rat. Peptides, 25 (10), 1783-1788. doi: 10.1016/j.peptides.2004.03.029 |
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2004 Journal Article Agonist-dependent internalization of the angiotensin II type one receptor (AT1): role of C-terminus phosphorylation in recruitment of β-arrestinsKule, Chris E., Karoor, Vijaya, Day, Jonathan N. E., Thomas, Walter G., Baker, Kenneth M., Dinh, Diem, Acker, Kathleen A. and Booz, George W. (2004). Agonist-dependent internalization of the angiotensin II type one receptor (AT1): role of C-terminus phosphorylation in recruitment of β-arrestins. Regulatory Peptides, 120 (1-3), 141-148. doi: 10.1016/j.regpep.2004.03.001 |
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2004 Journal Article Urotensin II: the old kid in townOnan, Döne, Hannan, Ross D. and Thomas, Walter G. (2004). Urotensin II: the old kid in town. Trends in Endocrinology and Metabolism, 15 (4), 175-182. doi: 10.1016/j.tem.2004.03.007 |
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2004 Journal Article Urotensin II Promotes Hypertrophy of Cardiac Myocytes via Mitogen-Activated Protein KinasesOnan, Done, Pipolo, Luisa, Yang, Eunice, Hannan, Ross D. and Thomas, Walter G. (2004). Urotensin II Promotes Hypertrophy of Cardiac Myocytes via Mitogen-Activated Protein Kinases. Molecular Endocrinology, 18 (9), 2344-2354. doi: 10.1210/me.2003-0309 |
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2004 Journal Article When 6 is 9: ‘Uncoupled’ AT 1 receptors turn signalling on its headThomas, W. G., Qian, H. and Smith, N. J. (2004). When 6 is 9: ‘Uncoupled’ AT 1 receptors turn signalling on its head. Cellular and Molecular Life Sciences, 61 (21), 2687-2694. doi: 10.1007/s00018-004-4245-2 |
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2003 Journal Article Direct Actions of Urotensin II on the Heart Implications for Cardiac Fibrosis and HypertrophyTzanidis, Alex, Hannan, Ross D., Thomas, Walter G., Onan, Döne, Autelitano, Dominic J., See, Fiona, Kelly, Darren J., Gilbert, Richard E. and Krum, Henry (2003). Direct Actions of Urotensin II on the Heart Implications for Cardiac Fibrosis and Hypertrophy. Circulation Research, 93 (3), 246-253. doi: 10.1161/01.RES.0000084382.64418.BC |
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2003 Journal Article Angiotensin receptors: form and function and distributionThomas, Walter G. and Mendelsohn, Frederick A. O. (2003). Angiotensin receptors: form and function and distribution. International journal of biochemistry & cell biology, 35 (6), 774-779. doi: 10.1016/S1357-2725(02)00263-7 |
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2003 Journal Article Adrenomedullin inhibits angiotensin AT1A receptor expression and function in cardiac fibroblastsAutelitano, Dominic J., Ridings, Rebecca, Pipolo, Luisa and Thomas, Walter G. (2003). Adrenomedullin inhibits angiotensin AT1A receptor expression and function in cardiac fibroblasts. Regulatory Peptides, 112 (1-3), 131-137. doi: 10.1016/S0167-0115(03)00031-4 |
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2003 Journal Article Arresting angiotensin type 1 receptorsThomas, Walter G. and Qian, Hongwei (2003). Arresting angiotensin type 1 receptors. Trends in Endocrinology & Metabolism, 14 (3), 130-136. doi: 10.1016/S1043-2760(03)00023-7 |
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2003 Journal Article Surface plasmon resonance spectroscopy in the study of membrane-mediated cell signallingMozsolits, Henriette, Thomas, Walter G. and Aguilar, Marie-Isabel (2003). Surface plasmon resonance spectroscopy in the study of membrane-mediated cell signalling. Journal of peptide science, 9 (2), 77-89. doi: 10.1002/psc.439 |
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2003 Journal Article Angiotensin II mediates cardiomyocyte hypertrophic growth pathways via MMP-dependent HB-EGF liberationSmith, Nicola J., Hannan, Ross D., Thomas, Walter G. and Lew, Rebecca A. (2003). Angiotensin II mediates cardiomyocyte hypertrophic growth pathways via MMP-dependent HB-EGF liberation. Letters in Peptide Science, 10 (5), 431-435. doi: 10.1007/s10989-004-2398-3 |
