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2016

Conference Publication

Hematopoietic Stem Cell Mobilization and Erythropoiesis Suppression in Response to Lipopolysaccharides Involve Two Distinct Tlr4-Depedent Mechanisms with Different Requirement for G-Csf Receptors

Bisht, Kavita, Jacobsen, Rebecca, Brunck, Marion, Keech, Thomas, McGirr, Crystal, Nowlan, Bianca, Barbier, Valerie, Winkler, Ingrid and Levesque, Jean-Pierre (2016). Hematopoietic Stem Cell Mobilization and Erythropoiesis Suppression in Response to Lipopolysaccharides Involve Two Distinct Tlr4-Depedent Mechanisms with Different Requirement for G-Csf Receptors. 45th Annual Scientific Meeting of the International-Society-for-Experimental-Hematology (ISEH), San Diego, CA, United States, 25-28 August 2016. Philadelphia, PA, United States: Elsevier. doi: 10.1016/j.exphem.2016.06.096

Hematopoietic Stem Cell Mobilization and Erythropoiesis Suppression in Response to Lipopolysaccharides Involve Two Distinct Tlr4-Depedent Mechanisms with Different Requirement for G-Csf Receptors

2016

Conference Publication

Suppression of medullar erythropoiesis in response to bacterial lipopolysaccharides (LPS) involves two distinct TLR4-dependent mechanisms with contrasted requirements for G-CSF receptors

Bisht, Kavita, Jacobsen, Rebecca, Nowlan, Bianca, McGirr, Crystal, Brunck, Marion, Keech, Thomas, Winkler, Ingrid G. and Levesque, Jean-Pierre (2016). Suppression of medullar erythropoiesis in response to bacterial lipopolysaccharides (LPS) involves two distinct TLR4-dependent mechanisms with contrasted requirements for G-CSF receptors. 58th Annual Meeting and Exposition of the American Society of Hematology (ASH), San Diego, CA, United States, 3-6 December 2016. Washington, DC, United States: American Society of Hematology. doi: 10.1182/blood.V128.22.546.546

Suppression of medullar erythropoiesis in response to bacterial lipopolysaccharides (LPS) involves two distinct TLR4-dependent mechanisms with contrasted requirements for G-CSF receptors

2015

Conference Publication

Carbon monoxide targets Notch1 and MAPK-ERK1/2 signaling pathways to block growth of lung carcinoma

Nemeth, Zsuzsanna, Csizmadia, Eva, Vikstrom, Lisa, Li, Mailin, Bisht, Kavita, Feizi, Alborz, Gallo, David, Otterbein, Leo, Fillinger, Janos, Dome, Balazs, B. Costa, Daniel and Wegiel, Barbara (2015). Carbon monoxide targets Notch1 and MAPK-ERK1/2 signaling pathways to block growth of lung carcinoma. AACR 106th Annual Meeting 2015, Philadelphia, PA, United States, 18-22 April 2015. Philadelphia, PA, United States: American Association for Cancer Research. doi: 10.1158/1538-7445.am2015-416

Carbon monoxide targets Notch1 and MAPK-ERK1/2 signaling pathways to block growth of lung carcinoma

2014

Journal Article

Biliverdin modulates the expression of C5aR in response to endotoxin in part via mTOR signaling

Bisht, Kavita, Wegiel, Barbara, Tampe, Jens, Neubauer, Oliver, Wagner, Karl-Heinz, Otterbein, Leo E. and Bulmer, Andrew C. (2014). Biliverdin modulates the expression of C5aR in response to endotoxin in part via mTOR signaling. Biochemical and Biophysical Research Communications, 449 (1), 94-99. doi: 10.1016/j.bbrc.2014.04.150

Biliverdin modulates the expression of C5aR in response to endotoxin in part via mTOR signaling

2014

Journal Article

Endogenous tetrapyrroles influence leukocyte responses to lipopolysaccharide in human blood: pre-clinical evidence demonstrating the anti-inflammatory potential of biliverdin

Bisht, Kavita, Tampe, Jens, Shing, Cecilia, Bakrania, Bhavisha, Winearls, James, Fraser, John, Wagner, Karl-Heinz and Bulmer, Andrew C. (2014). Endogenous tetrapyrroles influence leukocyte responses to lipopolysaccharide in human blood: pre-clinical evidence demonstrating the anti-inflammatory potential of biliverdin. Journal of Clinical and Cellular Immunology, 5 (3), 1000218. doi: 10.4172/2155-9899.1000218

Endogenous tetrapyrroles influence leukocyte responses to lipopolysaccharide in human blood: pre-clinical evidence demonstrating the anti-inflammatory potential of biliverdin

2012

Journal Article

Reduced circulating oxidized LDL is associated with hypocholesterolemia and enhanced thiol status in Gilbert syndrome

Boon, Ai-Ching, Hawkins, Clare L., Bisht, Kavita, Coombes, Jeff S., Bakrania, Bhavisha, Wagner, Karl-Heinz and Bulmer, Andrew C. (2012). Reduced circulating oxidized LDL is associated with hypocholesterolemia and enhanced thiol status in Gilbert syndrome. Free Radical Biology and Medicine, 52 (10), 2120-2127. doi: 10.1016/j.freeradbiomed.2012.03.002

Reduced circulating oxidized LDL is associated with hypocholesterolemia and enhanced thiol status in Gilbert syndrome

2012

Conference Publication

Reduced LDL oxidation is secondary to protection from in vivo thiol oxidation and hypocholesterolemia in Gilbert's syndrome

Bulmer, Andrew Cameron, Boon, Ai-Ching, Hawkins, Clare, Bisht, Kavita, Coombes, Jeff and Wagner, Karl-Heinz (2012). Reduced LDL oxidation is secondary to protection from in vivo thiol oxidation and hypocholesterolemia in Gilbert's syndrome. Experimental Biology Meeting, San Diego, United States, 21-25 April 2012. Bethesda, MD, United States: Federation of American Societies for Experimental Biology.

Reduced LDL oxidation is secondary to protection from in vivo thiol oxidation and hypocholesterolemia in Gilbert's syndrome

2010

Journal Article

Curcumin, resveratrol and flavonoids as anti-inflammatory, cyto- and DNA-protective dietary compounds

Bisht, Kavita, Wagner, Karl-Heinz and Bulmer, Andrew C. (2010). Curcumin, resveratrol and flavonoids as anti-inflammatory, cyto- and DNA-protective dietary compounds. Toxicology, 278 (1), 88-100. doi: 10.1016/j.tox.2009.11.008

Curcumin, resveratrol and flavonoids as anti-inflammatory, cyto- and DNA-protective dietary compounds

2009

Journal Article

Curcumin enhances non-inflammatory phagocytic activity of RAW264.7 cells

Bisht, Kavita, Choi, Woo H., Park, Shin Y., Chung, Moon K. and Koh, Woo S. (2009). Curcumin enhances non-inflammatory phagocytic activity of RAW264.7 cells. Biochemical and Biophysical Research Communications, 379 (2), 632-636. doi: 10.1016/j.bbrc.2008.12.135

Curcumin enhances non-inflammatory phagocytic activity of RAW264.7 cells