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2017 Journal Article The interplay of IKK, NF-κB and RIPK1 signaling in the regulation of cell death, tissue homeostasis and inflammationKondylis, Vangelis, Kumari, Snehlata, Vlantis, Katerina and Pasparakis, Manolis (2017). The interplay of IKK, NF-κB and RIPK1 signaling in the regulation of cell death, tissue homeostasis and inflammation. Immunological Reviews, 277 (1), 113-127. doi: 10.1111/imr.12550 |
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2017 Book Chapter Epithelial cell death and inflammation in skinKumari, Snehlata and Pasparakis, Manolis (2017). Epithelial cell death and inflammation in skin. Apoptotic and non-apoptotic cell death. (pp. 77-93) edited by Shigekazu Nagata and Hiroyasu Nakano. Cham, Switzerland: Springer. doi: 10.1007/82_2015_466 |
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2016 Journal Article RIPK1 counteracts ZBP1-mediated necroptosis to inhibit inflammationLin, Juan, Kumari, Snehlata, Kim, Chun, Van, Trieu-My, Wachsmuth, Laurens, Polykratis, Apostolos and Pasparakis, Manolis (2016). RIPK1 counteracts ZBP1-mediated necroptosis to inhibit inflammation. Nature, 540 (7631), 124-128. doi: 10.1038/nature20558 |
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2016 Conference Publication Role of RIP kinase signalling in the development of skin inflammation in mice with keratinocyte-specific IKK deficiencyKumari, S., Van, T. and Pasparakis, M. (2016). Role of RIP kinase signalling in the development of skin inflammation in mice with keratinocyte-specific IKK deficiency. Annual Meeting of the European-Society-for-Dermatological-Research (ESDR), Munich Germany, Sep 07-10, 2016. NEW YORK: ELSEVIER SCIENCE INC. doi: 10.1016/j.jid.2016.06.354 |
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2014 Journal Article Sharpin prevents skin inflammation by inhibiting TNFR1-induced keratinocyte apoptosisKumari, Snehlata, Redouane, Younes, Lopez-Mosqueda, Jaime, Shiraishi, Ryoko, Romanowska, Malgorzata, Lutzmayer, Stefan, Kuiper, Jan, Martinez, Conception, Dikic, Ivan, Pasparakis, Manolis and Ikeda, Fumiyo (2014). Sharpin prevents skin inflammation by inhibiting TNFR1-induced keratinocyte apoptosis. Elife, 3 e03422. doi: 10.7554/eLife.03422 |
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2014 Journal Article Epidermal RelA specifically restricts contact allergen-induced inflammation and apoptosis in skinKumari, Snehlata, Herzberg, Benjamin, Pofahl, Ruth, Krieg, Thomas and Haase, Ingo (2014). Epidermal RelA specifically restricts contact allergen-induced inflammation and apoptosis in skin. Journal of Investigative Dermatology, 134 (10), 2541-2550. doi: 10.1038/jid.2014.193 |
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2014 Journal Article RIPK1 maintains epithelial homeostasis by inhibiting apoptosis and necroptosisDannappel, Marius, Vlantis, Katerina, Kumari, Snehlata, Polykratis, Apostolos, Kim, Chun, Wachsmuth, Laurens, Eftychi, Christina, Lin, Juan, Corona, Teresa, Hermance, Nicole, Zelic, Matija, Kirsch, Petra, Basic, Marijana, Bleich, Andre, Kelliher, Michelle and Pasparakis, Manolis (2014). RIPK1 maintains epithelial homeostasis by inhibiting apoptosis and necroptosis. Nature, 513 (7516), 90-94. doi: 10.1038/nature13608 |
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2014 Conference Publication Altered tumor necrosis factor receptor signaling in keratinocytes triggers interleukin-24-dependent psoriasis-like skin inflammation in miceKumari, S., Bonnet, M., Witte, E., Wolk, K., Kollias, G., Sabat, R., Pasparakis, M. and Haase, I. (2014). Altered tumor necrosis factor receptor signaling in keratinocytes triggers interleukin-24-dependent psoriasis-like skin inflammation in mice. 41st Annual Meeting of the Arbeitsgemeinschaft-Dermatologische-Forschung (ADF), Cologne Germany, Mar 13-15, 2014. HOBOKEN: WILEY-BLACKWELL. |
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2013 Journal Article Tumor necrosis factor receptor signaling in keratinocytes triggers interleukin-24-dependent psoriasis-like skin inflammation in miceKumari, Snehlata, Bonnet, Marion C., Ulvmar, Maria H., Wolk, Kerstin, Karagianni, Niki, Witte, Ellen, Uthoff-Hachenberg, Claudia, Renauld, Jean-Christophe, Kollias, George, Toftgard, Rune, Sabat, Robert, Pasparakis, Manolis and Haase, Ingo (2013). Tumor necrosis factor receptor signaling in keratinocytes triggers interleukin-24-dependent psoriasis-like skin inflammation in mice. Immunity, 39 (5), 899-911. doi: 10.1016/j.immuni.2013.10.009 |
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2013 Conference Publication Epidermal RelA specifically restricts contact allergen induced inflammation and apoptosis in skinKumari, S., Krieg, T. and Haase, J. (2013). Epidermal RelA specifically restricts contact allergen induced inflammation and apoptosis in skin. International Investigative Dermatology Meeting, Edinburgh Scotland, May 08-11, 2013. NEW YORK: NATURE PUBLISHING GROUP. |
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2012 Conference Publication Epidermal control of psoriasis like skin inflammation by a TNF-alpha and IL-24 dependent mechanismKumari, S., Pasparakis, M., Sabat, R. and Haase, I. (2012). Epidermal control of psoriasis like skin inflammation by a TNF-alpha and IL-24 dependent mechanism. 75th Annual Meeting of the Society-for-Investigative-Dermatology, Raleigh Nc, May 09-12, 2012. NEW YORK: NATURE PUBLISHING GROUP. |
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2011 Conference Publication Control of psoriasis-like skin inflammation by epidermal keratinocytes through a tumour necrosis factor-alpha- and interleukin-24-dependent mechanismKumari, S. and Haase, I. (2011). Control of psoriasis-like skin inflammation by epidermal keratinocytes through a tumour necrosis factor-alpha- and interleukin-24-dependent mechanism. 6th International Congress of Psoriasis - from Gene to Clinic, London England, Dec 01-03, 2011. MALDEN: WILEY-BLACKWELL. |
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2011 Conference Publication Epidermal control of psoriasis like skin inflammation by a TNF-a and IL-24 dependent mechanismKumari, Snehlata and Haase, Ingo (2011). Epidermal control of psoriasis like skin inflammation by a TNF-a and IL-24 dependent mechanism. 41st Annual Meeting of the European-Society-for-Dermatological-Research, Barcelona Spain, Sep 07-10, 2011. NEW YORK: NATURE PUBLISHING GROUP. |
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2010 Conference Publication Systemic recruitment of myeloid cells upon deletion of IKK2 from epidermal keratinocytesKumari, Snehlata, Pasparakis, Manolis and Haase, Ingo (2010). Systemic recruitment of myeloid cells upon deletion of IKK2 from epidermal keratinocytes. 40th Annual Meeting of the European-Society-for-Dermatological-Research (ESDR 2010), Helsinki Finland, Sep 08-11, 2010. NEW YORK: NATURE PUBLISHING GROUP. |
