Honorary Professor Josephine Forbes
Honorary Professor

Josephine Forbes

Email: 

Availability

Honorary Professor Josephine Forbes is:
Available for supervision

Qualifications

  • Bachelor, University of Melbourne
  • Doctor of Philosophy, University of Melbourne

Search Professor Josephine Forbes’s works on UQ eSpace

259 works between 1995 and 2025
All (259) Journal Article (183) Book Chapter (5) Conference Publication (71)

101 - 120 of 259 works

2015

Book Chapter

Pathogenesis of diabetic microvascular complications

Forbes, Josephine M. and Harcourt, Brooke (2015). Pathogenesis of diabetic microvascular complications. International Textbook of Diabetes Mellitus. (pp. 873-888) wiley. doi: 10.1002/9781118387658.ch60

Pathogenesis of diabetic microvascular complications

2014

Journal Article

Glycemic control in diabetes is restored by therapeutic manipulation of cytokines that regulate beta cell stress

Hasnain, Sumaira Z., Borg, Danielle J., Harcourt, Brooke E., Tong, Hui, Sheng, Yonghua H., Ng, Choa Ping, Das, Indrajit, Wang, Ran, Chen, Alice C.-H., Loudovaris, Thomas, Kay, Thomas W., Thomas, Helen E., Whitehead, Jonathan P., Forbes, Josephine M., Prins, Johannes B. and McGuckin, Michael A. (2014). Glycemic control in diabetes is restored by therapeutic manipulation of cytokines that regulate beta cell stress. Nature Medicine, 20 (12), 1417-1426. doi: 10.1038/nm.3705

Glycemic control in diabetes is restored by therapeutic manipulation of cytokines that regulate beta cell stress

2014

Journal Article

Nox‐4 deletion reduces oxidative stress and injury by PKC‐α‐associated mechanisms in diabetic nephropathy

Thallas‐Bonke, Vicki, Jha, Jay C., Gray, Stephen P., Barit, David, Haller, Hermann, Schmidt, Harald H.H.W., Coughlan, Melinda T., Cooper, Mark E., Forbes, Josephine M. and Jandeleit‐Dahm, Karin A.M. (2014). Nox‐4 deletion reduces oxidative stress and injury by PKC‐α‐associated mechanisms in diabetic nephropathy. Physiological Reports, 2 (11) e12192, e12192.1-e12192.13. doi: 10.14814/phy2.12192

Nox‐4 deletion reduces oxidative stress and injury by PKC‐α‐associated mechanisms in diabetic nephropathy

2014

Conference Publication

Manipulation of dietary advanced glycation end-product content influences the progression of non-alcoholic fatty liver disease (NAFLD)

Leung, C., Herath, C. B., Zhiyuan, J., Leong, T., Forbes, J. M. and Angus, P. W. (2014). Manipulation of dietary advanced glycation end-product content influences the progression of non-alcoholic fatty liver disease (NAFLD). HOBOKEN: WILEY-BLACKWELL. doi: 10.1111/jgh.12794

Manipulation of dietary advanced glycation end-product content influences the progression of non-alcoholic fatty liver disease (NAFLD)

2014

Journal Article

Deletion of bone-marrow-derived receptor for AGEs (RAGE) improves renal function in an experimental mouse model of diabetes

Tesch, Greg, Sourris, Karly C., Summers, Shaun A., McCarthy, Domenica, Ward, Micheal S., Borg, Danielle J., Gallo, Linda A., Fotheringham, Amelia K., Pettit, Allison R., Yap, Felicia Y. T., Harcourt, Brooke E., Tan, Adeline L. Y., Kausman, Joshua Y., Nikolic-Paterson, David, Kitching, Arthur R. and Forbes, Josephine M. (2014). Deletion of bone-marrow-derived receptor for AGEs (RAGE) improves renal function in an experimental mouse model of diabetes. Diabetologia, 57 (9), 1977-1985. doi: 10.1007/s00125-014-3291-z

Deletion of bone-marrow-derived receptor for AGEs (RAGE) improves renal function in an experimental mouse model of diabetes

2014

Journal Article

Stress in the kidney is the road to pERdition: is endoplasmic reticulum stress a pathogenic mediator of diabetic nephropathy?

Zhuang, Aowen and Forbes, Josephine M. (2014). Stress in the kidney is the road to pERdition: is endoplasmic reticulum stress a pathogenic mediator of diabetic nephropathy?. Journal of Endocrinology, 222 (3), R97-R111. doi: 10.1530/JOE-13-0517

Stress in the kidney is the road to pERdition: is endoplasmic reticulum stress a pathogenic mediator of diabetic nephropathy?

2014

Journal Article

Ramipril inhibits AGE-RAGE-induced matrix metalloproteinase-2 activation in experimental diabetic nephropathy

Fukami, Kei, Yamagishi, Sho-ichi, Coughlan, Melinda T., Harcourt, Brooke E., Kantharidis, Phillip, Thallas-Bonke, Vicki, Okuda, Seiya, Cooper, Mark E. and Forbes, Josephine M. (2014). Ramipril inhibits AGE-RAGE-induced matrix metalloproteinase-2 activation in experimental diabetic nephropathy. Diabetology and Metabolic Syndrome, 6 (1) 86, 86. doi: 10.1186/1758-5996-6-86

Ramipril inhibits AGE-RAGE-induced matrix metalloproteinase-2 activation in experimental diabetic nephropathy

2014

Journal Article

SYNbiotics Easing Renal failure by improving Gut microbiologY (SYNERGY): a protocol of placebo-controlled randomised cross-over trial

Rossi, Megan, Johnson, David W., Morrison, Mark, Pascoe, Elaine, Coombes, Jeff S., Forbes, Josephine M., McWhinney, Brett C., Ungerer, Jacobus P. J., Dimeski, Goce and Campbell, Katrina L. (2014). SYNbiotics Easing Renal failure by improving Gut microbiologY (SYNERGY): a protocol of placebo-controlled randomised cross-over trial. BMC Nephrology, 15 (1) 106, 106.1-106.10. doi: 10.1186/1471-2369-15-106

SYNbiotics Easing Renal failure by improving Gut microbiologY (SYNERGY): a protocol of placebo-controlled randomised cross-over trial

2014

Journal Article

Circulating concentrations of soluble receptor for AGE are associated with age and AGER gene polymorphisms in children with newly diagnosed type 1 diabetes

Salonen, Kirsi M., Ryhanen, Samppa J., Forbes, Josephine M., Harkonen, Taina, Ilonen, Jorma, Laine, Antti-Pekka, Groop, Per-Henrik and Knip, Mikael (2014). Circulating concentrations of soluble receptor for AGE are associated with age and AGER gene polymorphisms in children with newly diagnosed type 1 diabetes. Diabetes Care, 37 (7), 1975-1981. doi: 10.2337/dc13-3049

Circulating concentrations of soluble receptor for AGE are associated with age and AGER gene polymorphisms in children with newly diagnosed type 1 diabetes

2014

Journal Article

Plasma advanced glycation end products (AGEs) and NF-κB activity are independent determinants of diastolic and pulse pressure

Sourris, Karly C., Lyons, Jasmine G., Dougherty, Sonia L., Chand, Vibhasha, Straznicky, Nora E., Schlaich, Markus P., Grima, Mariee T., Cooper, Mark E., Kingwell, Bronwyn A., de Courten, Maximilian P. J., Forbes, Josephine M. and de Courten, Barbora (2014). Plasma advanced glycation end products (AGEs) and NF-κB activity are independent determinants of diastolic and pulse pressure. Clinical Chemistry and Laboratory Medicine, 52 (1), 129-138. doi: 10.1515/cclm-2012-0850

Plasma advanced glycation end products (AGEs) and NF-κB activity are independent determinants of diastolic and pulse pressure

2014

Conference Publication

Flux of advanced glycation end products across the kidney as a contributor to renal disease in diabetes

Leung, C., Fotheringham, A. K., Zhuang, A., Borg, D. J., Ward, M. S., Coughlan, M. T., Mccarthy, D. A., Gallo, L. A., Harcourt, B. E. and Forbes, J. M. (2014). Flux of advanced glycation end products across the kidney as a contributor to renal disease in diabetes. 50th Annual Scientific Meeting of the Australian and New Zealand Society of Nephrology, Melbourne, Australia, 25–27 August 2014. Richmond Australia: Wiley-Blackwell Publishing Asia. doi: 10.1111/nep.12302

Flux of advanced glycation end products across the kidney as a contributor to renal disease in diabetes

2014

Conference Publication

Ager1 overexpression in glomerular podocytes results in renal disease which is exacerbated by diabetes

Zhuang, A., Sourris, K. C, Harcourt, B. E, Penfold, S. A., Fotheringham, A. K., McCarthy, D., Schulz, B. and Forbes, J. M. (2014). Ager1 overexpression in glomerular podocytes results in renal disease which is exacerbated by diabetes. 50th Annual Scientific Meeting of the Australian and New Zealand Society of Nephrology, Melbourne VIC, Australia, 25 - 27 August 2014. Richmond, VIC Australia: Wiley-Blackwell Publishing Asia. doi: 10.1111/nep.12301

Ager1 overexpression in glomerular podocytes results in renal disease which is exacerbated by diabetes

2014

Journal Article

Dietary glycotoxins exacerbate progression of experimental fatty liver disease

Leung, Christopher, Herath, Chandana B., Jia, Zhiyuan, Goodwin, Michelle, Mak, Kai Yan, Watt, Matthew J., Forbes, Josephine M. and Angus, Peter W. (2014). Dietary glycotoxins exacerbate progression of experimental fatty liver disease. Journal of Hepatology, 60 (4), 832-838. doi: 10.1016/j.jhep.2013.11.033

Dietary glycotoxins exacerbate progression of experimental fatty liver disease

2014

Conference Publication

Glyoxalase-1 Inhibition Leads to Podocyte Insulin Resistance and Features Resembling Diabetic Kidney Disease

Gallo, Linda A., Ward, Micheal S., Harcourt, Brooke E., Fotheringham, Amelia K., McCarthy, Domenica A., Penfold, Sally A. and Forbes, Josephine M. (2014). Glyoxalase-1 Inhibition Leads to Podocyte Insulin Resistance and Features Resembling Diabetic Kidney Disease. 14th Asian Pacific Congress of Nephrology, Tokyo, Japan, 14-17 May 2014. Richmond, VIC, Australia: Wiley-Blackwell. doi: 10.1111/nep.12236

Glyoxalase-1 Inhibition Leads to Podocyte Insulin Resistance and Features Resembling Diabetic Kidney Disease

2014

Conference Publication

Deficiency of rage in bone marrow cells reduces renal injury in diabetic mice

Tesch, G. H., Sourris, K. C., Summers, S. A., Mccarthy, D., Ward, M. S., Borg, D. J., Gallo, L. A., Fotheringham, A. K., Pettit, A., Yap, F. Y. T., Harcourt, B. E., Tan, A. L. Y., Kausman, J. Y., Nikolic-Paterson, D. J., Kitching, A. R. and Forbes, J. M. (2014). Deficiency of rage in bone marrow cells reduces renal injury in diabetic mice. 50th Annual Scientific Meeting of the Australian and New Zealand Society of Nephrology, Melbourne Australia, 25–27 August 2014. Richmond Australia: Wiley-Blackwell Publishing Asia. doi: 10.1111/nep.12301

Deficiency of rage in bone marrow cells reduces renal injury in diabetic mice

2014

Conference Publication

Mitochondrial targeting of superoxide by Mitoq improves metabolic renal dysfunction

Ward, M. S., Flemming, N. B., Gallo, L. A., Fotheringham, A., Gobe, G., Vesey, D. and Forbes, J. M. (2014). Mitochondrial targeting of superoxide by Mitoq improves metabolic renal dysfunction. 50th Annual Scientific Meeting of the Australian and New Zealand Society of Nephrology, Melbourne Australia, 25–27 August 2014. Richmond Australia: Wiley-Blackwell Publishing Asia. doi: 10.1111/nep.12301

Mitochondrial targeting of superoxide by Mitoq improves metabolic renal dysfunction

2013

Conference Publication

Report on ISN Forefronts, Melbourne, Australia, 4-7 October 2012: tubulointerstitial disease in diabetic nephropathy

Forbes, Josephine M., Harris, David C. H. and Cooper, Mark E. (2013). Report on ISN Forefronts, Melbourne, Australia, 4-7 October 2012: tubulointerstitial disease in diabetic nephropathy. New York, NY, United States: Elsevier. doi: 10.1038/ki.2013.89

Report on ISN Forefronts, Melbourne, Australia, 4-7 October 2012: tubulointerstitial disease in diabetic nephropathy

2013

Journal Article

Dietary advanced glycation end products as an environmental contributor to type 1 diabetes

Borg, Danielle J. and Forbes, Josephine M. (2013). Dietary advanced glycation end products as an environmental contributor to type 1 diabetes. IMARS Highlights, 8 (5), 21-22.

Dietary advanced glycation end products as an environmental contributor to type 1 diabetes

2013

Journal Article

Environmental determinants of islet autoimmunity (ENDIA): a pregnancy to early life cohort study in children at-risk of type 1 diabetes

Penno, Megan A. S., Couper, Jennifer J., Craig, Maria E., Colman, Peter G., Rawlinson, William D., Cotterill, Andrew M., Jones, Timothy W., Harrison, Leonard C., ENDIA Study Group and Forbes, Josephine M. (2013). Environmental determinants of islet autoimmunity (ENDIA): a pregnancy to early life cohort study in children at-risk of type 1 diabetes. BMC Pediatrics, 13 (1) 124, 124.1-124.15. doi: 10.1186/1471-2431-13-124

Environmental determinants of islet autoimmunity (ENDIA): a pregnancy to early life cohort study in children at-risk of type 1 diabetes

2013

Journal Article

Deficiency in mitochondrial complex I activity due to Ndufs6 gene trap insertion induces renal disease

Forbes, Josephine M., Ke, Bi-Xia, Tuong-Vi Nguyen, Henstridge, Darren C., Penfold, Sally A., Laskowski, Adrienne, Sourris, Karly C., Groschner, Lukas N., Cooper, Mark E., Thorburn, David R. and Coughlan, Melinda T. (2013). Deficiency in mitochondrial complex I activity due to Ndufs6 gene trap insertion induces renal disease. Antioxidants and Redox Signaling, 19 (4), 331-343. doi: 10.1089/ars.2012.4719

Deficiency in mitochondrial complex I activity due to Ndufs6 gene trap insertion induces renal disease

Current funding

  • 2024 - 2026
    Understanding RAGE Expression and Function in Human T and Dendritic Cells to Refine the Use of RAGE Targeting Therapeutics for Type 1 Diabetes Prevention
    Juvenile Diabetes Research Foundation - International
    Open grant
  • 2022 - 2026
    Designing and translating novel therapies for diabetes and kidney disease
    NHMRC Investigator Grants
    Open grant

Past funding

  • 2021 - 2024
    Innovative delivery methods for the biologic soluble RAGE to prevent Type 1 Diabetes
    JDRF Pilot and Innovation Awards
    Open grant
  • 2019 - 2021
    RAGE as a novel therapeutic target for T1D
    Juvenile Diabetes Research Foundation - International
    Open grant
  • 2019
    Dissolution DNP Hyperpolariser
    UQ Research Facilities Infrastructure Grants
    Open grant
  • 2019 - 2021
    Preventing progression of diabetic kidney disease by targeting mitochondrial function
    NHMRC Project Grant
    Open grant
  • 2016 - 2017
    Genetic susceptibility to mitochondrial dysfunction as a risk factor for diabetic nephropathy (DiaComp - NIH subaward administered by Augusta University)
    Augusta University
    Open grant
  • 2016 - 2018
    Advanced Glycation in the development of T1D (Helmsley Charitable Trust - George Eisenbath nPOD award for team science) - Administered by the University of Miami
    University of Miami
    Open grant
  • 2016 - 2020
    Investigating pathways to alleviate the burden of diabetes and kidney disease
    NHMRC Research Fellowship
    Open grant
  • 2016
    Mitochondrial dysfunction as a mediator of diabetic kidney disease
    Diabetes Australia Research Program
    Open grant
  • 2015 - 2018
    Environmental Determinants of Islet Autoimmunity (ENDIA) (University of Adelaide led JDRF Australian Type 1 Diabetes Clinical Research Network (T1DCRN) RFA )
    University of Adelaide
    Open grant
  • 2015 - 2017
    Kidney energetics in the development of nephropathy in type 1 diabetes (DiaComp - NIH subaward administered by Augusta University)
    Augusta University
    Open grant
  • 2015
    Glucose transport in the diabetic kidney
    Diabetes Australia Research Trust
    Open grant
  • 2015 - 2019
    IL-22 as a Suppressor of Pancreatic Beta-Cell Stress and a Treatment for Diabetes
    NHMRC Project Grant
    Open grant
  • 2014 - 2016
    Urinary metabolic profiles as predictors of early diabetic nephropathy
    Kidney Health Australia
    Open grant
  • 2013 - 2014
    Identification of a novel target for the treatment of diabetic kidney disease
    NHMRC Project Grant
    Open grant
  • 2013 - 2014
    Modern food processing as a contributor to type 1 diabetes
    NHMRC Project Grant
    Open grant
  • 2013 - 2015
    NHMRC Research Fellowship (SRF A): Delineating the role of advanced glycation in diabetes and nephropathy
    NHMRC Research Fellowship
    Open grant

Availability

Honorary Professor Josephine Forbes is:
Available for supervision

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Supervision history

Current supervision

Completed supervision

Enquiries

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