Associate Professor

Peter Noakes

Email: 
Phone: 
+61 7 336 51780
Phone: 
+61 7 336 51640

Availability

Associate Professor Peter Noakes is:
Available for supervision

Fields of research

Biological Sciences Immunology Neurosciences

Research interests

  • RESEARCH AREA 1:

    The cell and molecular mechanisms that underlie developmental motoneuron cell death. This involves examining a number of genetically modified mice that we and our colleagues have created. In each of these mice we have inactivated the genes that encode for synaptic signalling molecules, such as agrin and laminins, or molecules that are needed to cluster ion channels in the postsynaptic membranes of muscles and/or neurones, such as rapsyn and gephyrin.

  • RESEARCH AREA 2:

    The molecular mechanisms that underpin synapse formation and plasticity. This involves exploring the signal transduction mechanisms that are activated when signalling molecules such as neuregulin-1 (ARIA, beta-heregulin 1), or agrin active ErbB or MuSK receptors in the membrane of muscle respectively.

  • RESEARCH AREA 3:

    The biological role of P2X1, 2, 4 and 7 in the living animal. This is big project and involves making tissue inducible gene knockouts of these ion channels. At present we are well on the way to making such mice. However, before we can gain a proper understanding of their role in mice that are missing these ion channel subunits, we need to know their developmental expression profile in the developing mouse. In particular when and where they are expressed in the developing nervous system.

  • RESEARCH AREA 4:

    The biological role of TGFbeta 2 at adult synapses. This will involve looking at mice we are making where we the gene for TGFbeta is knockout out in adult muscle and/or in the adult spinal cord (a tissue inducible knockout out of TGFbeta 2). The mice should be ready sometime next year but no firm dates for this one.

  • RESEARCH AREA 5:

    The Control of synaptic gene expression. This project in the first part is simply a promoter and 3' UTR analysis of some 30 genes that appear at the same time in the postsynaptic region of skeletal muscle. It is aimed at looking for common regulatory elements and or spacing patterns of such elements (enhancers and repressors). It is high risk and involves some biological and mathematical knowledge. The biological part of this project will involve making transgenic mice with test non-coding DNA linked to reporter constructs such as green fluorescent protein (GFP). The aim here is to see if such elements can drive synapse expression of reporter genes (eg GFP)

  • RESEARCH AREA 6:

    The roles of insulin-like growth factors 1 and 2 (IGF-1, IGF-2), as well as insulin. in the proliferation and differentiation of mouse embryonic stem cells. This is not a neurobiology-based project, but is early developmental biology one. It will also have links through our USA collaborators to understanding the role of such growth factors in the proliferation tumor cells within the brain (U87 a nasty brain tumor). In particular, this project is aimed at looking at the signalling pathways that are activated when these growth factors activate their receptors, which are on these stem and tumor cells. We also want to know if these signalling pathways activate proliferation and differentiation. And if so what are the down stream target genes involved.

Search Professor Peter Noakes’s works on UQ eSpace

170 works between 1983 and 2024
All (170) Journal Article (106) Book Chapter (1) Conference Publication (63)

21 - 40 of 170 works

2019

Journal Article

Size-dependent vulnerability of lumbar motor neuron dendritic degeneration in SOD1(G93A) mice

Fogarty, Matthew J., Mu, Erica W. H., Lavidis, Nickolas A., Noakes, Peter G. and Bellingham, Mark C. (2019). Size-dependent vulnerability of lumbar motor neuron dendritic degeneration in SOD1(G93A) mice. The Anatomical Record, 303 (5) ar.24255, 1455-1471. doi: 10.1002/ar.24255

Size-dependent vulnerability of lumbar motor neuron dendritic degeneration in SOD1(G93A) mice

2019

Journal Article

The Role of Altered BDNF/TrkB Signaling in Amyotrophic Lateral Sclerosis

Pradhan, Jonu, Noakes, Peter G. and Bellingham, Mark C. (2019). The Role of Altered BDNF/TrkB Signaling in Amyotrophic Lateral Sclerosis. Frontiers in Cellular Neuroscience, 13 368, 368. doi: 10.3389/fncel.2019.00368

The Role of Altered BDNF/TrkB Signaling in Amyotrophic Lateral Sclerosis

2019

Journal Article

Revisiting the role of the innate immune complement system in ALS

Parker, Sandra E., Hanton, Angela M., Stefanou, Stephen N., Noakes, Peter G., Woodruff, Trent M. and Lee, John D. (2019). Revisiting the role of the innate immune complement system in ALS. Neurobiology of Disease, 127, 223-232. doi: 10.1016/j.nbd.2019.03.003

Revisiting the role of the innate immune complement system in ALS

2019

Journal Article

A seasonal comparison of the neuromuscular junction morphology of Bufo marinus

Ge, Dengyun, Noakes, Peter G. and Lavidis, Nickolas A. (2019). A seasonal comparison of the neuromuscular junction morphology of Bufo marinus. Journal of Comparative Neurology, 527 (12), 1931-1939. doi: 10.1002/cne.24661

A seasonal comparison of the neuromuscular junction morphology of Bufo marinus

2018

Journal Article

Complement components are upregulated and correlate with disease progression in the TDP-43 mouse model of amyotrophic lateral sclerosis

Lee, John D., Levin, Samantha C., Willis, Emily F., Li, Rui, Woodruff, Trent M. and Noakes, Peter G. (2018). Complement components are upregulated and correlate with disease progression in the TDP-43 mouse model of amyotrophic lateral sclerosis. Journal of Neuroinflammation, 15 (1) 171, 171. doi: 10.1186/s12974-018-1217-2

Complement components are upregulated and correlate with disease progression in the TDP-43 mouse model of amyotrophic lateral sclerosis

2018

Journal Article

Regulated alternative splicing of Drosophila Dscam2 is necessary for attaining the appropriate number of photoreceptor synapses

Kerwin, Sarah K., Li, Joshua Shing Shun, Noakes, Peter G., Shin, Grace Ji-eun and Millard, S. Sean (2018). Regulated alternative splicing of Drosophila Dscam2 is necessary for attaining the appropriate number of photoreceptor synapses. Genetics, 208 (2), 717-728. doi: 10.1534/genetics.117.300432

Regulated alternative splicing of Drosophila Dscam2 is necessary for attaining the appropriate number of photoreceptor synapses

2018

Journal Article

Defects in synaptic transmission at the neuromuscular junction precedes motor deficits in a TDP-43Q331K transgenic mouse model of amyotrophic lateral sclerosis

Chand, Kirat K., Lee, Kah Meng, Lee, John D., Qiu, Hao, Willis, Emily F., Lavidis, Nickolas A., Hilliard, Massimo A. and Noakes, Peter G. (2018). Defects in synaptic transmission at the neuromuscular junction precedes motor deficits in a TDP-43Q331K transgenic mouse model of amyotrophic lateral sclerosis. FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology, 32 (5), fj201700835R-2689. doi: 10.1096/fj.201700835R

Defects in synaptic transmission at the neuromuscular junction precedes motor deficits in a TDP-43Q331K transgenic mouse model of amyotrophic lateral sclerosis

2018

Conference Publication

IVT-14 Layer V pyramidal neurons showing synaptic hyper-excitability has altered TrkB receptor signaling in SOD1G3A mouse model of ALS

Pradhan, Jonu, Noakes, Peter G. and Bellingham, Mark C. (2018). IVT-14 Layer V pyramidal neurons showing synaptic hyper-excitability has altered TrkB receptor signaling in SOD1G3A mouse model of ALS. 29th International Symposium on ALS/MND, Glasgow, Scotland, 7-9 December 2018. Abingdon, Oxfordshire, United Kingdom: Taylor & Francis. doi: 10.1080/21678421.2018.1510569

IVT-14 Layer V pyramidal neurons showing synaptic hyper-excitability has altered TrkB receptor signaling in SOD1G3A mouse model of ALS

2017

Journal Article

Investigating methodological differences in the assessment of dendritic morphology of basolateral amygdala principal neurons – a comparison of Golgi-Cox and neurobiotin electroporation techniques

Klenowski, Paul M., Wright, Sophie E., Mu, Erica W.H., Noakes, Peter G., Lavidis, Nickolas A., Bartlett, Selena E., Bellingham, Mark C. and Fogarty, Matthew J. (2017). Investigating methodological differences in the assessment of dendritic morphology of basolateral amygdala principal neurons – a comparison of Golgi-Cox and neurobiotin electroporation techniques. Brain Sciences, 7 (12) 165, 1-12. doi: 10.3390/brainsci7120165

Investigating methodological differences in the assessment of dendritic morphology of basolateral amygdala principal neurons – a comparison of Golgi-Cox and neurobiotin electroporation techniques

2017

Journal Article

Motor Areas Show Altered Dendritic Structure in an Amyotrophic Lateral Sclerosis Mouse Model

Fogarty, Matthew J., Mu, Erica W. H., Lavidis, Nickolas A., Noakes, Peter G. and Bellingham, Mark C. (2017). Motor Areas Show Altered Dendritic Structure in an Amyotrophic Lateral Sclerosis Mouse Model. Frontiers in Neuroscience, 11 (NOV) 609, 1-16. doi: 10.3389/fnins.2017.00609

Motor Areas Show Altered Dendritic Structure in an Amyotrophic Lateral Sclerosis Mouse Model

2017

Journal Article

Cross-ethnic meta-analysis identifies association of the GPX3-TNIP1 locus with amyotrophic lateral sclerosis

Benyamin, Beben, He, Ji, Zhao, Qiongyi, Gratten, Jacob, Garton, Fleur, Leo, Paul J., Liu, Zhijun, Mangelsdorf, Marie, Al-Chalabi, Ammar, Anderson, Lisa, Butler, Timothy J., Chen, Lu, Chen, Xiang-Ding, Cremin, Katie, Deng, Hong-Weng, Devine, Matthew, Edson, Janette, Fifita, Jennifer A., Furlong, Sarah, Han, Ying-Ying, Harris, Jessica, Henders, Anjali K., Jeffree, Rosalind L., Jin, Zi-Bing, Li, Zhongshan, Li, Ting, Li, Mengmeng, Lin, Yong, Liu, Xiaolu ... Fan, Dongsheng (2017). Cross-ethnic meta-analysis identifies association of the GPX3-TNIP1 locus with amyotrophic lateral sclerosis. Nature Communications, 8 (1) 611, 611. doi: 10.1038/s41467-017-00471-1

Cross-ethnic meta-analysis identifies association of the GPX3-TNIP1 locus with amyotrophic lateral sclerosis

2017

Journal Article

Complement C5a-C5aR1 signalling drives skeletal muscle macrophage recruitment in the hSOD1G93A mouse model of amyotrophic lateral sclerosis

Wang, Haitao A., Lee, John D., Lee, Kah Meng, Woodruff, Trent M. and Noakes, Peter G. (2017). Complement C5a-C5aR1 signalling drives skeletal muscle macrophage recruitment in the hSOD1G93A mouse model of amyotrophic lateral sclerosis. Skeletal Muscle, 7 (1) 10, 10. doi: 10.1186/s13395-017-0128-8

Complement C5a-C5aR1 signalling drives skeletal muscle macrophage recruitment in the hSOD1G93A mouse model of amyotrophic lateral sclerosis

2017

Journal Article

Pharmacological inhibition of complement C5a-C5a1 receptor signalling ameliorates disease pathology in the hSOD1G93A mouse model of amyotrophic lateral sclerosis

Lee, John D., Kumar, Vinod, Fung, Jenny N. T., Ruitenberg, Marc J., Noakes, Peter G. and Woodruff, Trent M. (2017). Pharmacological inhibition of complement C5a-C5a1 receptor signalling ameliorates disease pathology in the hSOD1G93A mouse model of amyotrophic lateral sclerosis. British Journal of Pharmacology, 173 (8), 689-699. doi: 10.1111/bph.13730

Pharmacological inhibition of complement C5a-C5a1 receptor signalling ameliorates disease pathology in the hSOD1G93A mouse model of amyotrophic lateral sclerosis

2017

Journal Article

Functional decline at the aging neuromuscular junction is associated with altered laminin-α4 expression

Lee, Kah Meng, Chand, Kirat K., Hammond, Luke A., Lavidis, Nickolas A. and Noakes , Peter G. (2017). Functional decline at the aging neuromuscular junction is associated with altered laminin-α4 expression. Aging, 9 (3), 880-899. doi: 10.18632/aging.101198

Functional decline at the aging neuromuscular junction is associated with altered laminin-α4 expression

2017

Journal Article

Alterations in hypoglossal motor neurons due to GAD67 and VGAT deficiency in mice

Fogarty, Matthew J., Kanjhan, Refik, Yanagawa, Yuchio, Noakes, Peter G. and Bellingham, Mark C. (2017). Alterations in hypoglossal motor neurons due to GAD67 and VGAT deficiency in mice. Experimental Neurology, 289, 117-127. doi: 10.1016/j.expneurol.2016.12.004

Alterations in hypoglossal motor neurons due to GAD67 and VGAT deficiency in mice

2016

Journal Article

Loss of laminin-alpha4 results in pre- and postsynaptic modifications at the neuromuscular junction

Chand, Kirat K., Lee, Kah Meng, Lavidis, Nickolas A. and Noakes, P. G. (2016). Loss of laminin-alpha4 results in pre- and postsynaptic modifications at the neuromuscular junction. FASEB Journal, 31 (4), 1323-1336. doi: 10.1096/fj.201600899R

Loss of laminin-alpha4 results in pre- and postsynaptic modifications at the neuromuscular junction

2016

Journal Article

Cortical synaptic and dendritic spine abnormalities in a presymptomatic TDP-43 model of amyotrophic lateral sclerosis

Fogarty, Matthew J., Klenowski, Paul M., Lee, John D., Drieberg-Thompson, Joy R., Bartlett, Selena E., Ngo, Shyuan T., Hilliard, Massimo A., Bellingham, Mark C. and Noakes, Peter G. (2016). Cortical synaptic and dendritic spine abnormalities in a presymptomatic TDP-43 model of amyotrophic lateral sclerosis. Scientific Reports, 6 (1) 37968, 37968. doi: 10.1038/srep37968

Cortical synaptic and dendritic spine abnormalities in a presymptomatic TDP-43 model of amyotrophic lateral sclerosis

2016

Journal Article

Rats with a missense mutation in Atm display neuroinflammation and neurodegeneration subsequent to accumulation of cytosolic DNA following unrepaired DNA damage

Quek, Hazel, Luff, John, Cheung, KaGeen, Kozlov, Sergei, Gatei, Magtouf, Lee, C. Soon, Bellingham, Mark C., Noakes, Peter G., Lim, Yi Chieh, Barnett, Nigel L., Dingwall, Steven, Wolvetang, Ernst, Mashimo, Tomoji, Roberts, Tara L. and Lavin, Martin F. (2016). Rats with a missense mutation in Atm display neuroinflammation and neurodegeneration subsequent to accumulation of cytosolic DNA following unrepaired DNA damage. Journal of Leukocyte Biology, 101 (4), 927-947. doi: 10.1189/jlb.4VMA0716-316R

Rats with a missense mutation in Atm display neuroinflammation and neurodegeneration subsequent to accumulation of cytosolic DNA following unrepaired DNA damage

2016

Journal Article

A rat model of ataxia-telangiectasia: evidence for a neurodegenerative phenotype

Quek, Hazel, Luff, John, Cheung, KaGeen, Kozlov, Sergei, Gatei, Magtouf, Lee, C Soon, Bellingham, Mark C., Noakes, Peter G., Lim, Yi Chieh, Barnett, Nigel L., Dingwall, Steven, Wolvetang, Ernst, Mashimo, Tomoji, Roberts, Tara L. and Lavin, Martin F. (2016). A rat model of ataxia-telangiectasia: evidence for a neurodegenerative phenotype. Human Molecular Genetics, 26 (1), 109-123. doi: 10.1093/hmg/ddw371

A rat model of ataxia-telangiectasia: evidence for a neurodegenerative phenotype

2016

Journal Article

Marked changes in dendritic structure and spine density precede significant neuronal death in vulnerable cortical pyramidal neuron populations in the SOD1G93A mouse model of amyotrophic lateral sclerosis

Fogarty, Matthew J., Mu, Erica W. H., Noakes, Peter G., Lavidis, Nickolas A. and Bellingham, Mark C. (2016). Marked changes in dendritic structure and spine density precede significant neuronal death in vulnerable cortical pyramidal neuron populations in the SOD1G93A mouse model of amyotrophic lateral sclerosis. Acta Neuropathologica Communications, 4 (77) 77, 1-21. doi: 10.1186/s40478-016-0347-y

Marked changes in dendritic structure and spine density precede significant neuronal death in vulnerable cortical pyramidal neuron populations in the SOD1G93A mouse model of amyotrophic lateral sclerosis

Current funding

  • 2024 - 2027
    Mechanisms that promote the health of the motor unit in MND
    Cure for MND Foundation - Discovery Research Grants
    Open grant
  • 2022 - 2025
    Developing treatments for Duchenne Muscular Dystrophy
    Muscular Dystrophy Association
    Open grant

Past funding

  • 2023 - 2024
    Stabilizing Neuromuscular Signaling in Motor Neuron Disease
    Motor Neurone Disease Research Institute of Australia Inc Innovator Grant
    Open grant
  • 2020
    Molecular analyses of pre-synaptic compartments in diseased human and mouse brains
    Brain Foundation Research Gift
    Open grant
  • 2020 - 2023
    Fine tuning metabolic flux: Modulating substrate oxidation as a therapeutic strategy in motor neuron disease (MND)
    NHMRC IDEAS Grants
    Open grant
  • 2020 - 2023
    Towards the treatment of Duchenne Muscular Dystrophy
    NHMRC Development Grant
    Open grant
  • 2020 - 2024
    Understanding neuromuscular synaptic loss in Motor Neuron Disease
    NHMRC IDEAS Grants
    Open grant
  • 2018 - 2019
    Targeting the molecular basis of muscle weakness in motor neuron disease.
    Motor Neurone Disease Research Institute of Australia Inc
    Open grant
  • 2017 - 2018
    Targeting neuromuscular stability in motor neuron disease
    Motor Neurone Disease Research Institute of Australia Inc
    Open grant
  • 2016 - 2017
    Exploiting the opposing actions of complement receptors C3aR and C5aR1 in the treatment of MND
    Motor Neurone Disease Research Institute of Australia Inc
    Open grant
  • 2016
    A state-of-the-art facility for simulataneous photo-stimulation, high speed imaging and electrophysiological recording of multiple neurons in brain tissue and living organisms
    UQ Major Equipment and Infrastructure
    Open grant
  • 2015
    A state-of-the-art spinning disc confocal microscope for high speed imaging of live cells and super resolution microscopy
    UQ Major Equipment and Infrastructure
    Open grant
  • 2015
    The role of altered neuromuscular activity and mRNA transport in modifying the progression of Motor Neuron Disease
    Motor Neurone Disease Research Institute of Australia Inc
    Open grant
  • 2015
    The role of altered neuromuscular activity and mRNA transport in modifying the progression of Motor Neuron Disease
    Royal Brisbane and Women's Hospital Foundation
    Open grant
  • 2015
    The role of altered neuronal activity and mRNA transport in modifying the progression Motor Neuron Disease (MND)
    Royal Brisbane and Women's Hospital Foundation
    Open grant
  • 2014 - 2015
    The role of altered neuromuscular signaling in ALS: factors that modify the course of MND
    Motor Neurone Disease Research Institute of Australia Inc
    Open grant
  • 2014 - 2016
    The role of neuronal hyperactivity and neurotrophic factor signalling in synaptogenesis, dendrogenesis and neuron death in motor neuron disease
    NHMRC Project Grant
    Open grant
  • 2014 - 2016
    Understanding the role of TDP-43 in motor neuron disease.
    NHMRC Project Grant
    Open grant
  • 2012
    Establishment of Integrated Small Animal Metabolic and Physiology Assessment Facility
    UQ Major Equipment and Infrastructure
    Open grant
  • 2011
    Building UQ's analytical capacity in biomedical sciences
    UQ Major Equipment and Infrastructure
    Open grant
  • 2010
    Dual channel electrophysiological recordings
    NHMRC Equipment Grant
    Open grant
  • 2010
    Establishment of a digital Scanscope system for virtual microscopy
    UQ Major Equipment and Infrastructure
    Open grant
  • 2010
    In Situ Hybridization Suite for Biomedical Science Researchers
    NHMRC Equipment Grant
    Open grant
  • 2009 - 2012
    Development of a new method of motor unit number estimation for use in motor neurone disease
    NHMRC Project Grant
    Open grant
  • 2009 - 2012
    Molecular mechanisms that help organise effective synaptic transmission.
    NHMRC Project Grant
    Open grant
  • 2009
    Quantitative Histological Core Facility
    UQ School/Centre Co-Funding
    Open grant
  • 2009 - 2011
    The role of neuronal hyper-excitability in an animal model of motor neuron disease
    NHMRC Project Grant
    Open grant
  • 2008 - 2009
    The molecular basis of hyper-exactability in corticospinal neurons in a novel mouse model of amyotrophic lateral sclerosis
    Brain Foundation
    Open grant
  • 2008 - 2009
    High Speed Fluorescence Imaging coupled with Total Internal Reflection Microscopy and Fluorescence Recovery After Photobleaching System
    ARC Linkage Infrastructure, Equipment and Facilities
    Open grant
  • 2008
    Olympus disk-spinning confocal microscope system capable of simutaneously imaging a ratiometric (UV) non-ratiometric (visible range) dye
    Ramaciotti Foundation
    Open grant
  • 2008
    The molecular and functional basis of motor neuron hyper-excitability in an animal model of motor neuron disease.
    Motor Neurone Disease Research Institute of Australia Inc
    Open grant
  • 2008
    The role of synaptic laminins in the organisation of pre- and post-synaptic specializations at the neuromuscular synapse
    UQ External Support Enabling Grant
    Open grant
  • 2007 - 2009
    Evaluation of orally active anti-inflammatory C5a receptor antagonists in a transgenic rat motor neurone disease model
    NHMRC Project Grant
    Open grant
  • 2006 - 2008
    Phenotypic studies of the NaSi-1 transporter knock-out mouse
    NHMRC Project Grant
    Open grant
  • 2006 - 2008
    The role of central and peripheral synaptic activity in the developmental death of motoneurons.
    NHMRC Project Grant
    Open grant
  • 2005
    The effectiveness of C5a receptor antagonists in the treatment of motor neuron disease
    Motor Neurone Disease Research Institute of Australia Inc
    Open grant
  • 2003 - 2004
    A transgenic analysis of the physiologic roles of signaling domains in the growth hormone receptor
    NHMRC Project Grant
    Open grant
  • 2003
    The Role of TGFB2 In Maintaining the Function and Viability of Adult Motor Neurons
    Motor Neurone Disease Research Institute of Australia Inc
    Open grant
  • 2002
    Cell and molecular mechanisms that maintain and promote motoneuron survival: analysis of MuSK, agrin and rapsyn mutant lines of mice
    Ramaciotti Foundation
    Open grant
  • 2002 - 2004
    Role of synaptogenesis in development motoneuron cell death
    NHMRC Project Grant
    Open grant
  • 2000
    High-resolution, high quality digital time-lapse microscopy system
    NHMRC Equipment Grant
    Open grant
  • 1999 - 2001
    A Transgenic Analysis of the Basis for Growth Signalling by the Growth Hormone Receptor
    NHMRC Project Grant
    Open grant
  • 1999
    Functional analysis of neuromuscular synapses in mice that carry specific postsynaptic deletions
    Ramaciotti Foundation
    Open grant
  • 1999
    How neurotrophic factors modulate synaptic connections during development
    ARC Australian Research Council (Small grants)
    Open grant
  • 1999 - 2001
    Molecular Mechanisms Involved in the Formation, Maintenance and Viability of Synapses
    NHMRC Project Grant
    Open grant
  • 1999
    Molecular mechanisms of how motor nerve connections are maintained
    Motor Neurone Disease Research Institute of Australia Inc
    Open grant
  • 1999
    Network for brain research in mental disorders (Gene Targeting Consortium)
    Monash University
    Open grant
  • 1999
    Network for Brain Research into Mental Disorders (Gene Targeting Consortium)
    University of Melbourne
    Open grant
  • 1998 - 2000
    Synaptic Development in the Absence of á2-laminin, Agrin and Rapsyn
    NHMRC Project Grant
    Open grant
  • 1997
    Analysis of mutations affecting the formation and function of neuromuscular synapses
    NHMRC Project Grant
    Open grant
  • 1997
    Analysis of synapse formation in S/á2-laminin deficient mice
    University of Queensland New Staff Research Grant
    Open grant
  • 1997
    Neuronal cell death in the rapsyn deficient mouse
    UQ Foundation
    Open grant
  • 1997
    Pre-synaptic differentiation in S/beta2-Laminin deficient mice
    Ramaciotti Foundation
    Open grant

Availability

Associate Professor Peter Noakes is:
Available for supervision

Before you email them, read our advice on how to contact a supervisor.

Supervision history

Current supervision

Completed supervision

Enquiries

For media enquiries about Associate Professor Peter Noakes's areas of expertise, story ideas and help finding experts, contact our Media team:

communications@uq.edu.au