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2004

Journal Article

Synaptotagmin interaction with the syntaxin/SNAP-25 dimer is mediated by an evolutionarily conserved motif and is sensitive to inositol hexakisphosphate

Rickman, Colin, Archer, Deborah A., Meunier, Frederic A., Craxton, Molly, Fukuda, Mitsunori, Burgoyne, Robert D. and Davletov, Bazbek (2004). Synaptotagmin interaction with the syntaxin/SNAP-25 dimer is mediated by an evolutionarily conserved motif and is sensitive to inositol hexakisphosphate. Journal of Biological Chemistry, 279 (13), 12574-12579. doi: 10.1074/jbc.M310710200

Synaptotagmin interaction with the syntaxin/SNAP-25 dimer is mediated by an evolutionarily conserved motif and is sensitive to inositol hexakisphosphate

2004

Journal Article

High affinity interaction of syntaxin and SNAP-25 on the plasma membrane is abolished by botulinum toxin E

Rickman, Colin, Meunier, Frederic A., Binz, Thomas and Davletov, Bazbek (2004). High affinity interaction of syntaxin and SNAP-25 on the plasma membrane is abolished by botulinum toxin E. Journal of Biological Chemistry, 279 (1), 644-651. doi: 10.1074/jbc.M310879200

High affinity interaction of syntaxin and SNAP-25 on the plasma membrane is abolished by botulinum toxin E

2003

Journal Article

Dynamics of motor nerve terminal remodeling unveiled using SNARE-cleaving botulinum toxins: the extent and duration are dictated by the sites of SNAP-25 truncation

Meunier, Frederic, Lisk, Godfrey, Sesardic, Dorothea and Dolly, J. Oliver (2003). Dynamics of motor nerve terminal remodeling unveiled using SNARE-cleaving botulinum toxins: the extent and duration are dictated by the sites of SNAP-25 truncation. Molecular and Cellular Neuroscience, 22 (4), 454-466. doi: 10.1016/S1044-7431(02)00016-7

Dynamics of motor nerve terminal remodeling unveiled using SNARE-cleaving botulinum toxins: the extent and duration are dictated by the sites of SNAP-25 truncation

2003

Journal Article

Getting muscles moving again after botulinum toxin: novel therapeutic challenges

Foran, PG, Davletov, B and Meunier, FA (2003). Getting muscles moving again after botulinum toxin: novel therapeutic challenges. Trends In Molecular Medicine, 9 (7), 291-299. doi: 10.1016/S1471-4914(03)00113-8

Getting muscles moving again after botulinum toxin: novel therapeutic challenges

2003

Journal Article

Functional recycling of C2 domains throughout evolution: A comparative study of synaptotagmin, protein kinase C and phospholipase C by sequence, structural and modelling approaches

Jimenez, J. L., Smith, G. R., Contreras-Moreira, B., Sgouros, J. G., Meunier, F. A., Bates, P. A. and Schiavo, G. (2003). Functional recycling of C2 domains throughout evolution: A comparative study of synaptotagmin, protein kinase C and phospholipase C by sequence, structural and modelling approaches. Journal of Molecular Biology, 333 (3), 621-639. doi: 10.1016/j.jmb.2003.08.052

Functional recycling of C2 domains throughout evolution: A comparative study of synaptotagmin, protein kinase C and phospholipase C by sequence, structural and modelling approaches

2002

Journal Article

Botulinum neurotoxins: from paralysis to recovery of functional neuromuscular transmission

Meunier, FA, Schiavo, G and Molgo, J (2002). Botulinum neurotoxins: from paralysis to recovery of functional neuromuscular transmission. Journal of Physiology-paris, 96 (1-2), 105-113. doi: 10.1016/S0928-4257(01)00086-9

Botulinum neurotoxins: from paralysis to recovery of functional neuromuscular transmission

2002

Journal Article

Ionic mechanisms involved in the nodal swelling of myelinated axons caused by marine toxins

Benoit, Evelyne, Mattei, Cesar, Ouanounou, Gilles, Meunier, Frederic A., Suput, Dusan, Le Gall, Frederic, Marquais, Michel, Dechraoui, Marie Y. and Molgo, Jordi (2002). Ionic mechanisms involved in the nodal swelling of myelinated axons caused by marine toxins. Cellular & Molecular Biology Letters, 7 (2), 317-321.

Ionic mechanisms involved in the nodal swelling of myelinated axons caused by marine toxins

2002

Journal Article

Clostridial neurotoxins

Verastegui, C., Lalli, G., Bohnert, S., Meunier, F. A. and Schiavo, G. (2002). Clostridial neurotoxins. Journal of Toxicology-toxin Reviews, 21 (3), 203-227. doi: 10.1081/TXR-120014404

Clostridial neurotoxins

2002

Journal Article

Glycerotoxin from Glycera convoluta stimulates neurosecretion by up-regulating N-type Ca2+ channel activity

Meunier, Frédéric A., Feng, Zhong-Ping, Molgó, Jordi, Zamponi, Gerald W. and Schiavo, Giampietro (2002). Glycerotoxin from Glycera convoluta stimulates neurosecretion by up-regulating N-type Ca2+ channel activity. The EMBO Journal, 21 (24), 6733-6743. doi: 10.1093/emboj/cdf677

Glycerotoxin from Glycera convoluta stimulates neurosecretion by up-regulating N-type Ca2+ channel activity

2001

Journal Article

Phosphoinositides as key regulators of synaptic function

Osborne, Shona L., Meunier, Frédéric A. and Schiavo, Giampietro (2001). Phosphoinositides as key regulators of synaptic function. Neuron, 32 (1), 9-12. doi: 10.1016/S0896-6273(01)00455-X

Phosphoinositides as key regulators of synaptic function

2000

Journal Article

Latrophilin, neurexin, and their signaling-deficient mutants facilitate α-latrotoxin insertion into membranes but are not involved in pore formation

Volynski, KE, Meunier, FA, Lelianova, VG, Dudina, EE, Volkova, TM, Rahman, MA, Manser, C, Grishin, EV, Dolly, JO, Ashley, RH and Ushkaryov, YA (2000). Latrophilin, neurexin, and their signaling-deficient mutants facilitate α-latrotoxin insertion into membranes but are not involved in pore formation. Journal of Biological Chemistry, 275 (52), 41175-41183. doi: 10.1074/jbc.M005857200

Latrophilin, neurexin, and their signaling-deficient mutants facilitate α-latrotoxin insertion into membranes but are not involved in pore formation

2000

Journal Article

Ca2+ and Na+ contribute to the swelling of differentiated neuroblastoma cells induced by equinatoxin-II

Meunier, FA, Frangez, R, Benoit, E, Ouanounou, G, Rouzaire-Dubois, B, Suput, D and Molgo, J (2000). Ca2+ and Na+ contribute to the swelling of differentiated neuroblastoma cells induced by equinatoxin-II. Toxicon, 38 (11), 1547-1560. doi: 10.1016/S0041-0101(00)00088-X

Ca2+ and Na+ contribute to the swelling of differentiated neuroblastoma cells induced by equinatoxin-II

2000

Journal Article

Equinatoxin II increases intracellular Ca2+ in NG 108–15 cells

Frangež, Robert, Meunier, Frederick, Molgo, Jordi and Suput, Dusan (2000). Equinatoxin II increases intracellular Ca2+ in NG 108–15 cells. Pflugers Archiv European Journal of Physiology, 439 (7), R100-R101. doi: 10.1007/BF03376535

Equinatoxin II increases intracellular Ca2+ in NG 108–15 cells

2000

Journal Article

Trachynilysin mediates SNARE-dependent release of catecholamines from chromaffin cells via external and stored Ca2+

Meunier, FA, Mattei, C, Chameau, P, Lawrence, G, Colasante, C, Kreger, AS, Dolly, JO and Molgo, J (2000). Trachynilysin mediates SNARE-dependent release of catecholamines from chromaffin cells via external and stored Ca2+. Journal of Cell Science, 113 (7), 1119-1125.

Trachynilysin mediates SNARE-dependent release of catecholamines from chromaffin cells via external and stored Ca2+

2000

Journal Article

Effects of trachynilysin, a protein isolated from stonefish (Synanceia trachynis) venom, on frog atrial heart muscle

Sauviat, MP, Meunier, FA, Kreger, A and Molgo, J (2000). Effects of trachynilysin, a protein isolated from stonefish (Synanceia trachynis) venom, on frog atrial heart muscle. Toxicon, 38 (7), 945-959. doi: 10.1016/S0041-0101(99)00207-X

Effects of trachynilysin, a protein isolated from stonefish (Synanceia trachynis) venom, on frog atrial heart muscle

2000

Journal Article

Trachynilysin, a protein neurotoxin isolated from stonefish (Synanceia trachynis) venom, increases spontaneous quantal acetylcholine release from Torpedo marmorata neuromuscular junctions

Ouanounou, G, Mattei, C, Meunier, FA, Kreger, AS and Molgo, J (2000). Trachynilysin, a protein neurotoxin isolated from stonefish (Synanceia trachynis) venom, increases spontaneous quantal acetylcholine release from Torpedo marmorata neuromuscular junctions. Cybium, 24 (3), 149-156.

Trachynilysin, a protein neurotoxin isolated from stonefish (Synanceia trachynis) venom, increases spontaneous quantal acetylcholine release from Torpedo marmorata neuromuscular junctions

1999

Journal Article

Recreation of neuronal Kv1 channel oligomers by expression in mammalian cells using semliki forest virus

Shamotienko, O, Akhtar, S, Sidera, C, Meunier, FA, Ink, B, Weir, M and Dolly, JO (1999). Recreation of neuronal Kv1 channel oligomers by expression in mammalian cells using semliki forest virus. Biochemistry, 38 (51), 16766-16776. doi: 10.1021/bi991039n

Recreation of neuronal Kv1 channel oligomers by expression in mammalian cells using semliki forest virus

1999

Journal Article

Neurotoxins targetting receptor site 5 of voltage-dependent sodium channels increase the nodal volume of myelinated axons

Mattei, C, Dechraoui, MY, Molgo, J, Meunier, FA, Legrand, AM and Benoit, E (1999). Neurotoxins targetting receptor site 5 of voltage-dependent sodium channels increase the nodal volume of myelinated axons. Journal of Neuroscience Research, 55 (6), 666-673. doi: 10.1002/(SICI)1097-4547(19990315)55:63.0.CO;2-H

Neurotoxins targetting receptor site 5 of voltage-dependent sodium channels increase the nodal volume of myelinated axons

1999

Journal Article

Norepinephrine exocytosis stimulated by alpha-latrotoxin requires both external and stored Ca2+ and is mediated by latrophilin, G proteins and phospholipase C

Rahman, MA, Ashton, AC, Meunier, FA, Davletov, BA, Dolly, JO and Ushkaryov, YA (1999). Norepinephrine exocytosis stimulated by alpha-latrotoxin requires both external and stored Ca2+ and is mediated by latrophilin, G proteins and phospholipase C. Philosophical Transactions of the Royal Society of London Series B-Biological Sciences, 354 (1381), 379-386. doi: 10.1098/rstb.1999.0390

Norepinephrine exocytosis stimulated by alpha-latrotoxin requires both external and stored Ca2+ and is mediated by latrophilin, G proteins and phospholipase C

1998

Journal Article

Vesicle exocytosis stimulated by α-latrotoxin is mediated by latrophilin and requires both external and stored Ca2+

Davletov, BA, Meunier, FA, Ashton, AC, Matsushita, H, Hirst, WD, Lelianova, VG, Wilkin, GP, Dolly, JO and Ushkaryov, YA (1998). Vesicle exocytosis stimulated by α-latrotoxin is mediated by latrophilin and requires both external and stored Ca2+. EMBO Journal, 17 (14), 3909-3920. doi: 10.1093/emboj/17.14.3909

Vesicle exocytosis stimulated by α-latrotoxin is mediated by latrophilin and requires both external and stored Ca2+