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2007

Journal Article

An assessment of the role of SREBP 1 C in the pathogenesis of HCV-related steatosis

McPhersonS, Jonsson, JR, BarrieHD, CloustonAD and PowellEE (2007). An assessment of the role of SREBP 1 C in the pathogenesis of HCV-related steatosis. Hepatology, 46 (4), 438A-438A.

An assessment of the role of SREBP 1 C in the pathogenesis of HCV-related steatosis

2007

Journal Article

Progressive fibrosis in nonalcoholic steato hepatitis: Association with altered regeneration and a ductular reaction

Richardson M. M., Jonsson, J. R., Powell, E. E., Brunt, EM, Neuschwander-Tetri, BA, Bhathal, PS, Dixon, JB, Weltman, MD, Tilg, H, Moschen, AR, Purdie, DM, Demetris, AJ and Clouston, A. D. (2007). Progressive fibrosis in nonalcoholic steato hepatitis: Association with altered regeneration and a ductular reaction. Gastroenterology, 133 (1), 80-90. doi: 10.1053/j.gastro.2007.05.012

Progressive fibrosis in nonalcoholic steato hepatitis: Association with altered regeneration and a ductular reaction

2007

Journal Article

Progressive fibrosis in non-alcoholic steatohepatitis - Association with altered regeneration and a ductular reaction

RichardsonMM, Jonsson, JR, PowellEE, BruntEM, Neuschwander-TetriBA, BhathalPS, DixonJB, WeltmanMD, TilgH, MoschenAR, PurdieDM, DemetrisAJ and CloustonAD (2007). Progressive fibrosis in non-alcoholic steatohepatitis - Association with altered regeneration and a ductular reaction. Journal of Hepatology, 46, S49-S50. doi: 10.1016/S0168-8278(07)61711-5

Progressive fibrosis in non-alcoholic steatohepatitis - Association with altered regeneration and a ductular reaction

2007

Journal Article

An assessment of the role of endoplasmic reticulum (ER) stress in the pathogenesis of HCV

McPhersonS, PowellEE, BarrieHD, CloustonAD and Jonsson, JR (2007). An assessment of the role of endoplasmic reticulum (ER) stress in the pathogenesis of HCV. Hepatology, 46 (4), 444A-444A.

An assessment of the role of endoplasmic reticulum (ER) stress in the pathogenesis of HCV

2006

Journal Article

Non-response to antiviral therapy is associated with obesity and increased hepatic expression of suppressor of cytokine signalling 3 (SOCS-3) in patients with chronic hepatitis C, viral genotype 1

Walsh, MJ, Jonsson, JR, Richardson, MM, Lipka, GM, Purdie, DM, Clouston, D and Powell, EE (2006). Non-response to antiviral therapy is associated with obesity and increased hepatic expression of suppressor of cytokine signalling 3 (SOCS-3) in patients with chronic hepatitis C, viral genotype 1. Gut, 55 (4), 529-535. doi: 10.1136/gut.2005.069674

Non-response to antiviral therapy is associated with obesity and increased hepatic expression of suppressor of cytokine signalling 3 (SOCS-3) in patients with chronic hepatitis C, viral genotype 1

2006

Journal Article

Relationship between steatosis, inflammation, and fibrosis in chronic hepatitis C: A meta-analysis of individual patient data

Leandro, G, Mangia, A, Hui, J, Fabris, P, Rubbia-Brandt, L, Colloredo, G, Adinolfi, LE, Asselah, T, Jonsson, JR, Smedile, A, Terrault, N, Pazienza, V, Giordani, MT, Giostra, E, Sonzogni, A, Ruggiero, G, Marcellin, P, Powell, EE, George, J and Negro, F (2006). Relationship between steatosis, inflammation, and fibrosis in chronic hepatitis C: A meta-analysis of individual patient data. Gastroenterology, 130 (6), 1636-1642. doi: 10.1053/j.gastro.2006.03.014

Relationship between steatosis, inflammation, and fibrosis in chronic hepatitis C: A meta-analysis of individual patient data

2005

Journal Article

Steatosis is a cofactor in liver injury in hemochromatosis

Powell, Elizabeth E., Ali, Azmat, Clouston, Andrew D., Dixon, Jeannette L., Lincoln, Douglas J., Purdie, David M., Fletcher, Linda M., Powell, Lawrie W. and Jonsson, Julie R. (2005). Steatosis is a cofactor in liver injury in hemochromatosis. Gastroenterology, 129 (6), 1937-1943. doi: 10.1053/j.gastro.2005.09.015

Steatosis is a cofactor in liver injury in hemochromatosis

2005

Journal Article

Fibrosis correlates with a ductular reaction in hepatitis C: Roles of impaired replication, progenitor cells and steatosis

Clouston, Andrew D., Powell, Elizabeth E., Walsh, Meagan J., Richardson, Michelle M., Demetris, A. Jake and Jonsson, Julie R. (2005). Fibrosis correlates with a ductular reaction in hepatitis C: Roles of impaired replication, progenitor cells and steatosis. Hepatology, 41 (4), 809-818. doi: 10.1002/hep.20650

Fibrosis correlates with a ductular reaction in hepatitis C: Roles of impaired replication, progenitor cells and steatosis

2005

Journal Article

Steatosis: Co-factor in other liver diseases

Powell, EE, Jonsson, JR and Clouston, AD (2005). Steatosis: Co-factor in other liver diseases. Hepatology, 42 (1), 5-13. doi: 10.1002/hep.20750

Steatosis: Co-factor in other liver diseases

2005

Journal Article

Dangerous liaisons: The metabolic syndrome and nonalcoholic fatty liver disease

Powell, EE, Jonsson, JR and Clouston, AD (2005). Dangerous liaisons: The metabolic syndrome and nonalcoholic fatty liver disease. Annals of Internal Medicine, 143 (10), 753-754. doi: 10.7326/0003-4819-143-10-200511150-00015

Dangerous liaisons: The metabolic syndrome and nonalcoholic fatty liver disease

2005

Journal Article

Adiponectin and its receptors in patients with chronic hepatitis C

Jonsson, JR, Moschen, AR, Hickman, IJ, Richardson, MM, Kaser, S, Clouston, AD, Powell, EE and Tilg, H (2005). Adiponectin and its receptors in patients with chronic hepatitis C. Journal of Hepatology, 43 (6), 929-936. doi: 10.1016/j.jhep.2005.05.030

Adiponectin and its receptors in patients with chronic hepatitis C

2005

Conference Publication

Hepatic progenitor cell expansion and a periportal ductular reaction in NAFLD - Relationship with impaired hepatocyte proliferation and fibrosis, and reversal with treatment

Richardson, M., Jonsson, J., Powell, E., Brunt, E. M., Bhathal, P., Dixon, J. and Clouston, A. (2005). Hepatic progenitor cell expansion and a periportal ductular reaction in NAFLD - Relationship with impaired hepatocyte proliferation and fibrosis, and reversal with treatment. 56th Annual Meeting of the American Association for the Study of Liver Diseases, San Francisco, CA, United States, 11-15 November 2005. HOBOKEN: JOHN WILEY & SONS INC.

Hepatic progenitor cell expansion and a periportal ductular reaction in NAFLD - Relationship with impaired hepatocyte proliferation and fibrosis, and reversal with treatment

2005

Book Chapter

Does NASH or NAFLD contribute to comorbidity of other liver diseases?

Powell, Elizabeth E. (2005). Does NASH or NAFLD contribute to comorbidity of other liver diseases?. Fatty liver disease: NASH and related disorders. (pp. 276-288) edited by Geoffrey C. Farrell, Jacob George, Pauline de la M. Hall and Arthur J. McCullough. Oxford, United Kingdom: Blackwell.

Does NASH or NAFLD contribute to comorbidity of other liver diseases?

2005

Book Chapter

Does NASH or NAFLD contribute to comorbidity of other liver diseases?

Clouston, Andrew D. and Powell, Elizabeth E. (2005). Does NASH or NAFLD contribute to comorbidity of other liver diseases?. Fatty liver disease: NASH and related disorders. (pp. 276-288) edited by Geoffrey C. Farrell, Jacob George, Pauline de la M. Hall and Arthur J. McCullough. Malden, MA, United States: Blackwell Publishing. doi: 10.1002/9780470987438.ch23

Does NASH or NAFLD contribute to comorbidity of other liver diseases?

2005

Conference Publication

Obesity is associated with non-response to antiviral therapy and increased hepatic expression of SOCS-3 in chronic hepatitis C

Walsh, M.J., Jonsson, J.R., Richardson, M.M., Lipka, G.M., Purdie, D.M., Clouston, A.D. and Powell, E.E. (2005). Obesity is associated with non-response to antiviral therapy and increased hepatic expression of SOCS-3 in chronic hepatitis C. 40th Annual Meeting of the European Association for the Study of the Liver, Paris, France, 13-17 April 2005. Amsterdam , Netherlands: Elsevier.

Obesity is associated with non-response to antiviral therapy and increased hepatic expression of SOCS-3 in chronic hepatitis C

2005

Journal Article

A combination of genetic polymorphisms increases the risk of progressive disease in chronic hepatitis C

Richardson, M. M., Powell, E. E., Barrie, H. D., Clouston, A. D., Purdie, D. M. and Jonsson, J. R. (2005). A combination of genetic polymorphisms increases the risk of progressive disease in chronic hepatitis C. Journal of Medical Genetics, 42 (7), 1-6. doi: 10.1136/jmg.2005.032557

A combination of genetic polymorphisms increases the risk of progressive disease in chronic hepatitis C

2004

Journal Article

Nonalcoholic fatty liver disease: is all the fat bad?

Clouston, A. D. and Powell, E. E. (2004). Nonalcoholic fatty liver disease: is all the fat bad?. Internal Medicine Journal, 34 (4), 187-191. doi: 10.1111/j.1444-0903.2004.00574.x

Nonalcoholic fatty liver disease: is all the fat bad?

2004

Journal Article

Increased mononuclear cell activation and apoptosis early after human liver transplantation is associated with a reduced frequency of acute rejection

Jonsson, Julie R., Gu, Wenyi, Vanags, Daina M., Bishop, G. Alex, McCaughan, Geoffrey W., Fawcett, Jonathon, Lynch, Stephen V., Balderson, Glenda A., Powell, Elizabeth E. and Clouston, Andrew D. (2004). Increased mononuclear cell activation and apoptosis early after human liver transplantation is associated with a reduced frequency of acute rejection. Liver Transplantation, 10 (3), 397-403. doi: 10.1002/lt.20084

Increased mononuclear cell activation and apoptosis early after human liver transplantation is associated with a reduced frequency of acute rejection

2004

Journal Article

Bone mineral density in Australian children, adolescents and adults with cystic fibrosis: a controlled cross sectional study

Buntain, HM, Greer, RM, Schluter, PJ, Wong, JCH, Batch, JA, Potter, JM, Lewindon, PJ, Powell, E, Wainwright, CE and Bell, SC (2004). Bone mineral density in Australian children, adolescents and adults with cystic fibrosis: a controlled cross sectional study. Thorax, 59 (2), 149-155. doi: 10.1136/thorax.2003.006726

Bone mineral density in Australian children, adolescents and adults with cystic fibrosis: a controlled cross sectional study

2004

Journal Article

CCR5-Delta 32 mutation is strongly associated with primary sclerosing cholangitis

Eri, R, Jonsson, JR, Pandeya, N, Purdie, DM, Clouston, AD, Martin, N, Duffy, D, Powell, EE, Fawcett, J, Florin, THJ and Radford-Smith, GL (2004). CCR5-Delta 32 mutation is strongly associated with primary sclerosing cholangitis. Genes And Immunity, 5 (6), 444-450. doi: 10.1038/sj.gene.6364113

CCR5-Delta 32 mutation is strongly associated with primary sclerosing cholangitis