Chronic inflammatory demyelinating polyradiculoneuropathy and severe peripheral oedema: a renal explanation

Early pregnancy factor suppresses experimental autoimmune encephalomyelitis induced in Lewis rats with myelin basic protein and in SJL/J mice with myelin proteolipid protein peptide 139-151

Surges of Increased T Cell Reactivity to an Encephalitogenic Region of Myelin Proteolipid Protein Occur More Often in Patients with Multiple Sclerosis than in Healthy Subjects

Effects of Cyclosporin A Treatment on Clinical Course and Inflammatory Cell Apoptosis in Experimental Autoimmune Encephalomyelitis Induced in Lewis Rats by Inoculation with Myelin Basic Protein

Cytokine Expression by Inflammatory Cells Obtained from the Spinal Cords of Lewis Rats with Experimental Autoimmune Encephalomyelitis Induced by Inoculation with Myelin Basic Protein and Adjuvants

Microglia are more susceptible than macrophages to apoptosis in the central nervous system in experimental autoimmune Encephalomyelitis through a mechanism not involving Fas (CD95)

The roles of Fas, Fas Ligand and Bcl-2 in T Cell apoptosis in the central nervous system in experimental autoimmune Encephalomyelitis

Increased Immunoreactivity to Two Overlapping Peptides of Myelin Proteolipid Protein in Multiple Sclerosis

Corrigendum to "A Study Of Human T-Cell Lines Generated From Multiple Sclerosis Patients And Controls By Stimulation With Peptides Of Myelin Basic Protein" (J. Neuroimmunol. 70, 65-74)

Increased apoptosis of T lymphocytes and macrophages in the central and peripheral nervous systems of Lewis rats with experimental autoimmune encephalomyelitis treated with dexamethasone

Erratum: A study of human T-cell lines generated from multiple sclerosis patients and controls by stimulation with peptides of myelin basic protein (J. Neuroimmunol. 70, 65-74 (1996))

Corticosteroid Treatment Of Experimental Autoimmune Encephalomyelitis In The Lewis Rat Results in Loss of V Beta 8.2+ and Myelin Basic Protein-Reactive Cells from the Spinal Cord, with Increased Total T-Cell Apoptosis but Reduced Apoptosis of V Beta 8.2+

A Study Of Human T-Cell Lines Generated From Multiple Sclerosis Patients And Controls By Stimulation With Peptides Of Myelin Basic Protein

Apoptosis of Vβ8.2+ T lymphocytes in the spinal cord during recovery from experimental autoimmune encephalomyelitis induced in Lewis rats by inoculation with myelin basic protein

Conduction abnormalities are restricted to the central nervous system in experimental autoimmune encephalomyelitis induced by inoculation with proteolipid protein but not with myelin basic protein (Vol 117, pg 975, 1994)

The Proximal Peripheral Nervous System Is A Major Site Of Demyelination In Experimental Autoimmune Encephalomyelitis Induced In The Lewis Rat By A Myelin Basic Protein-Specific T Cell

Restoration of conduction in the spinal roots correlates with clinical recovery from experimental autoimmune encephalomyelitis

Antigen-Specific Down-Regulation of Myelin Basic Protein-Reactive T Cells During Spontaneous Recovery From Experimental Autoimmune Encephalomyelitis: Further Evidence of Apoptotic Deletion of Autoreactive T Cells in the Central Nervous System

Clinical And Histological Findings in Proteolipid Protein-Induced Experimental Autoimmune Encephalomyelitis (EAE) In The Lewis Rat. Distribution of Demyelination Differs From That in EAE Induced by Other Antigens

Macrophage apoptosis in the central nervous system in experimental autoimmune encephalomyelitis