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Dr Carmen Mathmann
Dr

Carmen Mathmann

Email: 
Phone: 
+61 7 344 36970

Overview

Background

My research focuses on understanding the cellular and molecular mechanisms that regulate innate immune responses during infection and inflammation. I have expertise in host–pathogen interactions, macrophage biology, Toll-like receptor signalling, and intracellular trafficking, with a particular interest in how spatiotemporal regulation of immune receptors shapes antimicrobial and inflammatory responses. Through the integration of cell biology, immunology, and advanced imaging approaches, my work aims to identify mechanisms that can be leveraged for host-directed therapies against infectious diseases, including tuberculosis and bacterial infections. My research program combines fundamental discovery science with translational approaches to improve our understanding of immune regulation in health and disease.

Availability

Dr Carmen Mathmann is:
Available for supervision

Qualifications

  • Doctor of Philosophy of Cellular Biology, Columbia University in the City of New York

Research interests

  • Innate Immune Signalling

    Investigating the molecular mechanisms that regulate innate immune responses during infection and inflammation. My research focuses on Toll-like receptor signalling, receptor trafficking, and the spatial regulation of signalling complexes that control antimicrobial and inflammatory responses. This work aims to identify pathways that can be targeted to improve immune function and disease outcomes.

  • Host–Pathogen Interactions

    Studying how immune cells detect and respond to bacterial pathogens, with a particular focus on macrophage responses to intracellular infection. My work examines the mechanisms that influence pathogen clearance, immune activation, and host defence, with the goal of informing new therapeutic strategies for infectious diseases.

  • Translational Immunology

    Applying mechanistic insights from immunology to develop clinically relevant approaches for infectious and inflammatory diseases. My research integrates cellular immunology, functional immune screening, and collaborative translational studies to identify biomarkers and therapeutic targets that can improve patient outcomes.

Research impacts

My research aims to improve our understanding of how the immune system responds to infectious diseases and how these responses can be harnessed to develop better treatments. By investigating the cellular mechanisms that regulate inflammation and host defence, my work has identified new insights into Toll-like receptor signalling and immune pathways that influence the body's ability to fight bacterial infections.

Research outcomes include:

  • Competitive research funding from national and international organisations, including the American Association of Immunologists, Queensland Government, Translational Research Institute, and Metro South Health.
  • Publications in leading peer-reviewed journals that advance understanding of innate immune signalling and host–pathogen interactions.
  • Collaborative projects focused on translating immunology research into improved approaches for the prevention, diagnosis and treatment of infectious diseases.
  • Supervision and mentoring of Honours, postgraduate and early-career researchers, helping build future capacity in biomedical and immunology research.

Works

Search Professor Carmen Mathmann’s works on UQ eSpace

8 works between 2011 and 2025

1 - 8 of 8 works

2025

Journal Article

TLR4 endocytosis and endosomal TLR4 signaling are distinct and independent outcomes of TLR4 activation

Schultz, Thomas E., Mathmann, Carmen D., Domínguez Cadena, Leslie C., Muusse, Timothy W., Kim, Hyoyoung, Wells, James W., Ulett, Glen C., Hamerman, Jessica A., Brooks, Andrew J., Kobe, Bostjan, Sweet, Matthew J., Stacey, Katryn J. and Blumenthal, Antje (2025). TLR4 endocytosis and endosomal TLR4 signaling are distinct and independent outcomes of TLR4 activation. EMBO Reports, 26 (10) 2250056, 2740-2766. doi: 10.1038/s44319-025-00444-2

TLR4 endocytosis and endosomal TLR4 signaling are distinct and independent outcomes of TLR4 activation

2024

Journal Article

Myddosomes in Toll-like receptor signaling-one to bind and rule them all

Mathmann, Carmen D., Schultz, Thomas E., Cadena, Leslie C. Dominguez and Blumenthal, Antje (2024). Myddosomes in Toll-like receptor signaling-one to bind and rule them all. Immunology and Cell Biology, 102 (9), 752-756. doi: 10.1111/imcb.12816

Myddosomes in Toll-like receptor signaling-one to bind and rule them all

2023

Conference Publication

Spatiotemporal regulation of rp105 subcellular localization shapes endosomal TLR4 signaling

Mathmann, Carmen, Schultz, Thomas, Ongtengco, Cherica Felize J., Dalton, Emma, Domínguez Cadena, Leslie C., Zamoshnikova, Alina, Stow, Jennifer L. and Blumenthal, Antje (2023). Spatiotemporal regulation of rp105 subcellular localization shapes endosomal TLR4 signaling. IMMUNOLOGY 2023 Meeting, Washington, DC, United States, 11 - 15 May 2023. Rockville, MD, United States: American Association of Immunologists. doi: 10.4049/jimmunol.210.supp.160.06

Spatiotemporal regulation of rp105 subcellular localization shapes endosomal TLR4 signaling

2022

Journal Article

Inhibition of the master regulator of Listeria monocytogenes virulence enables bacterial clearance from spacious replication vacuoles in infected macrophages

Tran, Thao Thanh, Mathmann, Carmen D., Gatica-Andrades, Marcela, Rollo, Rachel F., Oelker, Melanie, Ljungberg, Johanna K., Nguyen, Tam T. K., Zamoshnikova, Alina, Kummari, Lalith K., Wyer, Orry J. K., Irvine, Katharine M., Melo-Bolívar, Javier, Gross, Annette, Brown, Darren, Mak, Jeffrey Y. W., Fairlie, David P., Hansford, Karl A., Cooper, Matthew A., Giri, Rabina, Schreiber, Veronika, Joseph, Shannon R., Simpson, Fiona, Barnett, Timothy C., Johansson, Jörgen, Dankers, Wendy, Harris, James, Wells, Timothy J., Kapetanovic, Ronan, Sweet, Matthew J. ... Blumenthal, Antje (2022). Inhibition of the master regulator of Listeria monocytogenes virulence enables bacterial clearance from spacious replication vacuoles in infected macrophages. PLoS Pathogens, 18 (1) e1010166, e1010166. doi: 10.1371/journal.ppat.1010166

Inhibition of the master regulator of Listeria monocytogenes virulence enables bacterial clearance from spacious replication vacuoles in infected macrophages

2021

Journal Article

Rab6b localizes to the Golgi complex in murine macrophages and promotes tumor necrosis factor release in response to mycobacterial infection

Domínguez Cadena, Leslie C., Schultz, Thomas E., Zamoshnikova, Alina, Donovan, Meg L., Mathmann, Carmen D., Yu, Chien-Hsiung, Mori, Giorgia, Stow, Jennifer L and Blumenthal, Antje (2021). Rab6b localizes to the Golgi complex in murine macrophages and promotes tumor necrosis factor release in response to mycobacterial infection. Immunology and Cell Biology, 99 (10), 1067-1076. doi: 10.1111/imcb.12503

Rab6b localizes to the Golgi complex in murine macrophages and promotes tumor necrosis factor release in response to mycobacterial infection

2019

Journal Article

A tension-independent mechanism reduces Aurora B-mediated phosphorylation upon microtubule capture by CENP-E at the kinetochore

Taveras, Carmen, Liu, Chenshu and Mao, Yinghui (2019). A tension-independent mechanism reduces Aurora B-mediated phosphorylation upon microtubule capture by CENP-E at the kinetochore. Cell Cycle, 18 (12), 1349-1363. doi: 10.1080/15384101.2019.1617615

A tension-independent mechanism reduces Aurora B-mediated phosphorylation upon microtubule capture by CENP-E at the kinetochore

2016

Journal Article

Meeting report - New York Symposium on Quantitative Biology of the Cell

Liu, Chenshu, Taveras, Carmen, Kulukian, Anita, Ma, Rui, Ezratty, Ellen and Mao, Yinghui (2016). Meeting report - New York Symposium on Quantitative Biology of the Cell. Journal of Cell Science, 129 (8), 1525-1529. doi: 10.1242/jcs.188375

Meeting report - New York Symposium on Quantitative Biology of the Cell

2011

Journal Article

p53 inhibits mRNA 3' processing through its interaction with the CstF/BARD1 complex

Nazeer, F. I., Devany, E., Mohammed, S., Fonseca, D., Akukwe, B., Taveras, C. and Kleiman, F. E. (2011). p53 inhibits mRNA 3' processing through its interaction with the CstF/BARD1 complex. Oncogene, 30 (27), 3073-3083. doi: 10.1038/onc.2011.29

p53 inhibits mRNA 3' processing through its interaction with the CstF/BARD1 complex

Funding

Current funding

  • 2024 - 2027
    Improving tuberculosis diagnosis and care through functional immune screening
    TRI Leading Innovations through New Collaborations Scheme
    Open grant

Past funding

  • 2022 - 2023
    Molecular regulators of inflammation
    The American Association of Immunologists Careers in Immunology Fellowship
    Open grant
  • 2020 - 2021
    Dr Carmen Mathmann - AQ WRAP
    Advance Queensland Women's Research Assistance Program
    Open grant
  • 2019 - 2020
    Spatio-temporal regulation of TLR signaling
    The American Association of Immunologists Career Reentry Fellowship
    Open grant

Supervision

Availability

Dr Carmen Mathmann is:
Available for supervision

Looking for a supervisor? Read our advice on how to choose a supervisor.

Supervision history

Current supervision

  • Doctor Philosophy

    Molecular regulators of macrophage functions during Mycobacterium tuberculosis infection

    Associate Advisor

    Other advisors: Professor Antje Blumenthal

Media

Enquiries

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